《成人急诊医学一览》.(Adult.Emergency.Medicine.at.a.Glance).(英)托马斯·休斯&杰森·克里克山克.文字版.pdf
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《成人急诊医学一览》主要面向医学院的学生和刚刚开始工作的医生,主要内容为临床医学和急救。易理解,重实际,要点全面,可供复习参考。此书作为一般大众可能稍有距离,但是作为医学专业的学生来说,则极为重要精简。整本书配有47页的全彩图像,环节清楚、简洁。值得一读。
Following the familiar, easy-to-use at a Glance format, and in full-colour, this brand new title provides an accessible introduction and revision aid for medical students and junior doctors. Reflecting the increased profile of Emergency Medicine in clinical practice and the medical school curriculum, Adult Emergency Medicine at a Glance provides a user-friendly overview of the key subjects that will enable any student or junior doctor to 'hit the ground running' when they enter one of the most exciting areas of clinical medicine.
Adult Emergency Medicine at a Glance is:
A concise, visually orientated course in emergency medicine that is perfect for both study and revision.
Organised around symptoms: 'Short of Breath', rather than diagnoses: 'Pneumonia'.
Focused on the most common or dangerous conditions you will see in the Emergency Department and includes the latest cardiac resuscitation guidelines.
Comprehensively illustrated throughout with over 47 full-page colour illustrations.
作者介绍:
Thomas Hughes is Consultant Emergency Physician, John Radcliffe Hospital, Oxford, and Honorary Senior Lecturer in Emergency Medicine, University of Oxford
Jaycen Cruickshank is Director of Emergency Medicine, Ballarat Health Services, and Senior Lecturer in Emergency Medicine, University of Melbourne, Victoria, Australia
Adult Emergency Medicine at a GlanceAdult Emergency
Medicine at a Glance
Thomas Hughes
Bt.MSC, MBA, MRCP, FRCS, FCEM
Consultant Emergency Physician
John Radcliffe Hospital, Oxford
Honorary Senior Lecturer in Emergency Medicine
University of Oxford
Jaycen Cruickshank
MCR, FACEM
Director of Emergency Medicine
Ballarat Health Services, Victoria, Australia
Senior Lecturer in Emergency Medicine
University of Melbourne, Victoria, Australia
A John Wiley Sons, Inc., PublicationThis edition ? rst published 2011, ? 2011 by Thomas Hughes and Jaycen Cruickshank
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Library of Congress Cataloging-in-Publication Data
Hughes, Thomas, MSc.
Adult emergency medicine at a glance Thomas Hughes, Jaycen Cruickshank.
p. ; cm.
Includes index.
ISBN 978-1-4051-8901-9
1. Emergency medicine--Handbooks, manuals, etc. I. Cruickshank, Jaycen. II. Title.
[DNLM: 1. Emergency Medicine--methods--Handbooks. 2. Adult. WB 105 H894a 2011]
RC86.8.H84 2011
616.02'5--dc22
2010024551
ISBN: 9781405189019
A catalogue record for this book is available from the British Library.
Set in 9 on 11.5 pt Times by Toppan Best-set Premedia Limited
1 2011Contents 5
Contents
Preface and acknowledgements 7
List of abbreviations 8
1 Life in the Emergency Department 10
2 Diagnosis 12
3 Shock and intravenous ? uids 14
4 Imaging in the Emergency Department 16
5 Analgesia 18
6 Airway management and sedation 20
7 Blood gas analysis 22
8 Trauma: primary survey 24
9 Trauma: secondary survey 26
10 Major head and neck injury 28
11 Minor head and neck injury 30
12 Wounds 32
13 Burns 34
14 Hand injuries 36
15 Wrist and forearm injuries 38
16 Shoulder and elbow injuries 40
17 Back pain, hip and knee injuries 42
18 Tibia, ankle and foot injuries 44
19 Abdominal pain 46
20 Urology problems 48
21 Ear, nose, throat and dental problems 50
22 Eye problems 52
23 Obstetrics and gynaecology problems 54
24 Toxicology: general principles 56
25 Toxicology: speci? c poisons 58
26 Psychiatry: self-harm and capacity 60
27 Psychiatry: the disturbed patient 62
28 Observational medicine 64
29 Loss of function and independence 66
30 Syncope, collapse and falls 68
31 Slow heart rate 70
32 Fast heart rate 72
33 Cardiac arrest 74
34 Chest pain: cardiovascular 76
35 Chest pain: non-cardiovascular 78
36 Shortness of breath 80
37 Anaphylaxis 82
38 Sepsis 84
39 Endocrine emergencies 86
40 Gastroenterology 88
41 Headache 90
42 Stroke and transient ischaemic attack 92
43 Seizures 94
44 Hypothermia and hyperthermia 96
45 Pre-hospital medicine 98
46 Major incident 100
47 Chemical, biological, radiation, nuclear and explosive
incidents 102
Case studies: questions 104
Case studies: answers 107
Index 113Preface and Acknowledgements 7
Preface
Emergency Medicine has undergone a quiet revolution over the
past twenty years due to a variety of factors that have changed the
way medicine is practiced.
· Increasing demand and expectations of medical care.
· Reduction of junior doctors hours.
· An ageing population.
· Fragmentation of out of hours care.
· Reduced hospital bed - stay.
· Sub - specialisation of inpatient medical and surgical practice.
· Litigation.
These factors have pushed expert decision - making towards
the front door of the hospital so that the correct diagnosis and
treatment start as soon as possible in the patient s journey. As
other specialties have moved away from the acute assessment and
treatment of patients, Emergency Medicine has expanded to ? ll
the vacuum left, and in doing so has increased its realm of practice
substantially.
Emergency Medicine is exciting and confronting, intimidating
and liberating – it is the chance to exercise and hone your diag-
nostic and practical skills in a well - supervised environment.
Clinical staff who work in the ED have all been through the
inevitable feelings of fear, uncertainty and doubt that come
with the territory, and want you to experience the enjoyment
and satisfaction of working in an area of medicine that is never
boring.
When trainees start Emergency Medicine, it is often the ? rst
time they have seen patients before any other staff. To use a tra-
ditional analogy, they have seen plenty of needles, and may be very
good at recognising them, but now they are faced with haystacks,in which may be hidden a variety of sharp shiny objects.
Medical textbooks usually describe topics by anatomy or pathol-
ogy (needles), e.g. heart failure, which tends to assume the diag-
nostic process. In this book we have tried to organise topics by
presentation (haystacks), e.g. ‘ short of breath , and have tried to
articulate the key features that help us ? nd the needles.
We are both great fans of the ‘ At a Glance series, and have
enjoyed the challenge of combining the breadth of practice of adult
Emergency Medicine with the concise nature of the ‘ At a Glance
format. We hope you enjoy this book and ? nd it useful as you
explore this most dynamic area of medicine.
Acknowledgements
We would like to thank Karen Moore and Laura Murphy at
Wiley - Blackwell for their support and advice (and let s face it,patience) during the elephantine gestation of the book. Also Helen
Harvey for understanding deadlines, and Jane Fallows for doing
such a great job with the illustrations. We thank the students of
Oxford and Melbourne Universities, whose inquiring minds keep
us on our toes, and whose questions stimulated us to think of new
ways to present the information we have used here. We are both
lucky enough to have worked with an exceptional colleague, Philip
Catterson, whose teamwork, leadership and hard work have
helped us to achieve success in our jobs, and from whom we have
learned the few interpersonal skills we have.
In addition TH would like to thank: Professor Christopher
Bulstrode who has been an inspiration and mentor and without
whose support the book would not have happened. My family,and particularly my wife Marina for her support. My work col-
leagues for continuing to tolerate me most of the time, and Jackie
and Tracey, the ED secretaries who keep me in order. Nic Weir,Rob Janas and David Bowden for their help in sourcing images
and the ? nal preparation.
JC would like to thank: All the people who have taught me
along the way, particularly Trevor Jackson and Steven Pincus. My
parents Ron and Christine for making everything possible, and my
wife Kerry and sons Jesse and Flynn for making everything
worthwhile.
Thomas Hughes
Jaycen Cruickshank 8 List of abbreviations
List of abbreviations
ED Emergency Department
EDTA ethylene diamine tetraacetate
ELISA enzyme - linked immunosorbent assay
ENT ear, nose and throat
EPL extensor pollicis longus
ESR erythrocyte sedimentation rate
ETT endotracheal tube
FAST acronym for focused abdominal sonography in trauma;
also face, arm, speech, time to call ambulance
FB foreign body
FBCFBE full blood countexamination
FiO 2 fraction of inspired of oxygen (as %)
FFP fresh frozen plasma
FOOSH fall onto an outstretched hand
GA general anaesthetic
GA β HS group A β - haemolytic Streptococcus
GCS Glasgow Coma ScaleScore
GI gastrointestinal
GP general practitioner
H1N1 swine ? u virus
H5N1 avian ? u virus
HbA 1c glycated (glycosylated) haemoglobin
HCO3--
bicarbonate ion
hCG human chorionic gonadotrophin
HDU high dependency unit
HHS hyperosmolar hyperglycaemic state
HIV human immunode? ciency virus
HOCM hypertrophic obstructive cardiomyopathy
HONK hyperosmolar non - ketotic acidosis
HR heart rate
HVZ herpes varicella zoster
IBS irritable bowel syndrome
ICP intracranial pressure
ICU intensive care unit
IgE immunoglobulin E
IVDU intravenous drug use
IVF in vitro fertilisation
IVRA intravenous regional anaesthesia
IVU intravenous urogram
JVP jugular venous pressure
KUB kidneys, ureters and bladder
LA local anaesthetic
LCL lateral collateral ligament of knee
LFT liver function test
LMP last menstrual period
LNMP last normal menstrual period
LOC loss of consciousness
LP lumbar puncture
LR likelihood ratio
LRTI lower respiratory tract infection
MAOI monoamine oxidase inhibitor
MAP mean arterial pressure
MCL medial collateral ligament of knee
MCPJ metacarpophalangeal joint
MDI metered dose inhaler
AAA abdominal aortic aneurysm
ABC airway, breathing, circulation
ABCD
2
acronym to assess stroke risk in a patient with TIA
ABCDE airway, breathing, circulation, disability, exposure
ABG arterial blood gases
ACE angiotensin - converting enzyme
ACh acetylcholine
ACJ acromioclavicular joint
ACL anterior cruciate ligament of knee
ACS acute coronary syndrome
ACTH adrenocorticotrophic hormone
AD aortic dissection
AF atrial ? brillation
AIDS acquired immunode? ciency syndrome
AMT4 four-point abbreviated mental test score
AP antero - posterior
APL abductor pollicis longus
AV arteriovenous; also atrioventricular
AVN atrioventricular node
AXR abdominal X - ray
BDZ benzodiazepine
BP blood pressure
bpm beats per minute
CAGE acronym for alcohol screening questions
CAP community - acquired pneumonia
cAMP cyclic adenonsine monophosphate
CBRNE chemical, biological, radiological, nuclear,explosive
CK creatine kinase
CNS central nervous system
CO carbon monoxide
COHb carboxyhaemoglobin
COPD chronic obstructive pulmonary disease
CPAP continuous positive airway pressure
CPP cerebral perfusion pressure
CPR cardiopulmonary resuscitation
CRAO central retinal artery occlusion
CRP C - reactive protein
CRVO central retinal vein occlusion
CT computed tomography
CTPA CT pulmonary angiography
CURB - 65 confusion, urea, respiratory rate, blood pressure,age over 65 (acronym for pneumonia severity
factors)
CVP central venous pressure
CXR chest X - ray; also unit for X - ray dose, 1 CXR ≈ 3
days background radiation
DIPJ distal interphalangeal joint
DKA diabetic ketoacidosis
DM diabetes mellitus
DSH deliberate self - harm
DUMBELS diarrhoea, urination, miosis, bronchorrhoeabron-
chospasm, emesis, lacrimation, salivation (acronym
for clinical effects of organophosphate poisoning)
DVT deep vein thrombosis List of abbreviations 9
MI myocardial infarction
MR magnetic resonance
N 2 O nitrous oxide
NAC N - acetylcysteine
NICE National Institute for Health and Clinical Excellence
NIV non - invasive ventilation
NNT number needed to treat
NNH number needed to harm
NoF fractured neck of femur
NSAID non - steroidal anti - in? ammatory drug
NSTEMI non - ST segment elevation myocardial infarction
OD overdose
OP organophosphate
OPG oral pantomogram
ORIF open reduction and internal ? xation
PA postero - anterior
PCL posterior cruciate ligament of knee
PE pulmonary embolism
PEA pulseless electrical activity
PEF peak expiratory ? ow
PEFR peak expiratory ? ow rate
PID pelvic in? ammatory disease
PPCI primary percutaneous coronary intervention
PPI proton pump inhibitor
PPM permanent pacemaker
PR per rectum
PT prothrombin time
PV per vaginam
RA regional anaesthesia
RBBB right bundle branch block
RoSC return of spontaneous circulation
SAH subarachnoid haemorrhage
SAN sinoatrial node
SARS severe acute respiratory syndrome
SDH subdural haematoma
SoB short(ness) of breath
SOCRATES acronym for pain history
SOL space - occupying lesion
SSRI selective serotonin reuptake inhibitor
STD sexually transmitted disease
STEMI ST segment elevation myocardial infarction
STI sexually transmitted infection
SVT supraventricular tachycardia
TBSA total body surface area
TCA tricyclic antidepressant
TFT thyroid function test
TIA transient ischaemic attack
TIMI thrombolysis in myocardial infarction
TMT tarsometatarsal
tPA tissue plasminogen activator
U + E urea and electrolytes
UA unstable angina
URTI upper respiratory tract infection
UTI urinary tract infection
VBG venous blood gases
VF ventricular ? brillation
VQ ventilationperfusion
VT ventricular tachycardia
VVS vasovagal syncope
WCC white cell count 10 Adult Emergency Medicine at a Glance, 1st edition. ? Thomas Hughes and Jaycen Cruickshank. Published 2011 by Blackwell Publishing Ltd.
1 Life in the Emergency Department
Patients with acute health needs
GP
Triage
1
Resus
2
High
acuity
– time
critical
3
High
acuity
– not time
critical
4
Low
acuity
5
Non-urgent
Community
ED
Discharged patients
Short stay
wards
Community
ED
Hospital
Cough
Inpatient
units
This chapter describes the way the Emergency Department oper-
ates, and some of the unwritten rules. The prevalence of Emergency
Department - based drama generates plenty of misconceptions
about what occurs in the Emergency Department. For instance,it is generally inadvisable to say ‘ stat at the end of one s
sentences, and neither of the authors has been mistaken for George
Clooney!
What h appens w hen a p atient a rrives
at the Emergency Department?
Alert p hone
Also known as the ‘ red phone or sometimes ‘ the Bat - phone , this
is the dedicated phone line that the ambulance service uses to pre -
warn the Emergency Department of incoming patients likely to
need resuscitation.
Triage
The concept of triage comes from military medicine – doing the
most good for the most people. This ensures the most effective use
of limited resources, and that the most unwell patients are seen
· rst.
Nurses rather than doctors are usually used to perform the
triage because doctors tend to start treating patients. Systems
of rapid assessment and early treatment by senior medical staff
can be effective, but risk diverting attention from the most ill
patients. Life in the Emergency Department 11
Reception r egistration
The reception staff are essential to the function of the Emergency
Department: they register patients on the hospital computer
system, source old notes and keep an eye on the waiting room.
They have to deal with dif? cult and demanding patients, and are
good at spotting the sick or deteriorating patient in the waiting
room.
Waiting r oom
Adult and paediatric patients should have separate waiting rooms,and some sort of entertainment is a good idea. Aggression and
dissatisfaction in waiting patients has been largely eliminated in
the UK by the 4 - hour standard of care: all patients must be
seen and discharged from the Emergency Department within 4
hours.
Treatment a reas in the
Emergency Department
Resuscitation b ays
Resuscitation bays are used for critically ill and unstable patients
with potentially life - threatening illness. They have advanced moni-
toring facilities, and plenty of space around the patient for clinical
staff to perform procedures. X - rays can be performed within this
area.
High a cuity a rea
This is the area where patients who are unwell or injured, but who
do not need a resuscitation bay, are managed. Medical conditions
and elderly patients with falls are common presentations in this
area.
Low a cuity a rea
The ‘ walking wounded – patients with non - life - threatening
wounds and limb injuries – are seen here. Patients with minor
illness are discouraged from coming to the Emergency Department,but continue to do so for a variety of reasons.
There is a common misconception that patients in this area are
similar to general practice or family medicine patients. Numerous
studies have found that there is an admission rate of about 5% and
an appreciable mortality in low acuity patients, whereas only
about 1% of GP consultations result in immediate hospital
admission.
Other a reas
Imaging
Imaging, such as X - rays and ultrasound, are integral to Emergency
Department function. Larger Emergency Departments have their
own CT scanner.
Relatives r oom
When dealing with the relatives of a critically ill patient and break-
ing bad news, doctors and relatives need a quiet area where infor-
mation is communicated and digested. This room needs to be close
to the resuscitation area.
Observationshort stay w ard
This is a ward area close to the Emergency Department, run by
Emergency Department staff. This unit treats patients who would
otherwise need hospital admission for a short time, to enable them
to be fully stabilised and assessed. The function of these units is
described in Chapter 28 .
Hospital in the h ome
Some hospitals run a ‘ hospital in the home programme for
patients who do not need to be in hospital but who need
certain therapy, e.g. intravenous antibiotics, anticoagulation. The
Emergency Department is the natural interface between home
and hospital.
Culture of the Emergency Department
There is a much ? atter (less hierarchical) organisational structure in
the Emergency Department than most other areas in the hospital.
This occurs because all levels of medical, nursing and other staff
work together all the time, and the department cannot function
without their cooperation. Ensuring good teamwork requires good
leadership, an atmosphere of mutual respect and a bit of patience
and understanding.
The resulting atmosphere can be one of the most enjoyable and
satisfying places to work in a hospital. A feature of this less hier-
archical culture that surprises junior doctors is that nurses will
question their decisions; this is a sign of a healthy culture in which
errors are less likely to occur, and is actively encouraged.
Emergency Department r ules
Being a doctor in the Emergency Department is different from
elsewhere in the hospital. There is nowhere to hide and, for
the ? rst time in most medical careers, you are responsible for
making the decisions. On the positive side, there are plenty
of people around to help you, who have all been through the
same process.
Some basic advice:
· Write legible, timed, dated notes.
· Show respect for other professional groups and be prepared to
learn from them.
· Do not be late for your shifts; do not call in sick less than 6 hours
before a shift.
· Patients who re - present are high risk and need senior review.
· Take your breaks. You need them.
· Keep calm.
· If in doubt, ask.
· Do not pick up so many patients that you cannot keep track of
them.
· Do not avoid work or avoid seeing dif? cult patients. We do notice.
· The nurse in charge is usually right.
With so many people working closely together in a stressful
atmosphere, it is inevitable that con? icts will occur. Do not
let them fester; some ground rules for resolving such con? icts
are:
· Resolve it now.
· Do it in private.
· Do it face to face.
· Focus on facts.
· Criticise action, not person.
· Agree why it is important.
· Agree on a remedy.
· Finish on a positive.
12 Adult Emergency Medicine at a Glance, 1st edition. ? Thomas Hughes and Jaycen Cruickshank. Published 2011 by Blackwell Publishing Ltd.
2 Diagnosis
10
1
0.1
ODDS 1:20
A type of d-dimer test has a specificity of 50% and a sensitivity of 95%:
this allows you to calculate the likelihood ratios to rule a condition in (+LR)
or out (–LR)
A woman presents with a history suggestive of DVT, and her
probability of having a DVT is low; about 5%, according to the
Wells score (see Ch. 35).
The d-dimer test comes back negative.
What is the chance she has a DVT?
Probability 5%
0.1:20 = 1:200
0.5%
0.1
1 – Sn 1 – 0.95
1 – Sp
= 1.9 Sn +LR =
1 – 0.5
0.95 = = 0.1 –LR =
Sn 0.5
=
10
1 x
x
=
x
x
=
x
x
=
0.1
ODDS 1:20
If the d-dimer test had come back positive, what is the chance she
has a DVT?
Probability 5%
1.9:20 = 1:10
10%
1.9
The result (0.5%) is the risk of missing a DVT in this patient.
Bearing in mind that no test is perfect, a result below 1% is
generally taken as an acceptable level of risk
10
1
0.1
x
x
=
10
1
0.1
ODDS 1:1
Probability 50%
0.1:1 = 1:10
10%
0.1 5 ODDS 1:100
Probability 1%
5:100 = 1:20
5%
Despite the positive d-dimer, this patient only has a 10% chance
of having a DVT
This shows that this d-dimer test is useful for excluding a DVT in a low-risk population, but that a positive test does not mean a DVT is present
A patient who has been immobile following a recent operation
for cancer has pain in his lower leg. You assess his risk of
DVT as about 50%, but the test comes back negative.
What is the probability he has a DVT?
Despite a negative d-dimer, he still has a 10% chance of having
a DVT. D-dimer cannot ‘rule out’ DVT in a high risk patient
A young man with mild suprapubic pain, whom you estimate has
a 1% chance of having a UTI, has a positive urine leucocyte test
(+ LR = 5) on dipstick testing. What is the chance he has a UTI?
Despite a positive test with a fairly high + LR he still only has a 5%
chance of having a UTI because the condition was so unlikely
in the first place
The Emergency Department is a diagnosis machine, taking
people with a wide variety of symptoms, labelling them with
a diagnosis, treating and then discharging them whenever
safe to do so. There are plenty of opportunities for this process
to go wrong, and it is important to understand how this can
occur.
‘ If you listen carefully to the patient, they will tell you the
diagnosis . W Osler
Despite a couple of thousand years of medical education, we are
still not really sure how the diagnostic process works.
· Some people work forward from history, examination and a
shortlist of differential diagnoses.
· Others work backwards from a list of likely diagnoses to weight
these according to symptoms and examination.
· Experts make their diagnoses by pattern recognition.
It may be that expertise occurs with the development of cogni-
tive ? exibility to use multiple diagnostic strategies to integrate and
test the result. Using t ests
In the past, the Emergency Department used a few simple tests
to inform decision - making. An X - ray of an injured limb has a
binary outcome: broken not broken. As technology and the scope
of Emergency Medicine has increased, the tests have become more
numerous and less black and white, and there is a need to ration-
alise and manage the uncertainty generated.
The most common way of describing a test s performance is to
use sensitivity and speci? city .
Confusion m atrix
Diagnosis 13
SpIN – a very Speci? c test rules a condition IN .
SnOUT – a very Sensitive test rules a condition OUT .
Bayes theorem
The chance of something being true or false depends not only
on the quality of the test that one is performing, but impor-
tantly , how likely the event is in the ? rst place.
Thomas Bayes, an eighteenth - century English priest, deduced
the principles that underpin the way we use tests in medicine:
pre - t est p robability × likelihood r atio = post - t est
p robability
To calculate this, we use odds (like horse racing odds) rather
than probability, but the two are obviously very closely related.
The likelihood ratio is calculated from the test s sensitivity
(Sn) and speci? city (Sp) and is a much better measure of a test s
clinical usefulness in ruling a disease in (positive LR) or out
(negative LR).
+=
·
·=
·
LR Sn
Sp
LR Sn
Sp 1
1
How d o I d e? ne p re - t est p robability?
The triage process uses an expert nurse to assess clinical status
and is an assessment of the probability of serious illness. The
fact that a patient has arrived at the Emergency Department at
all, rather than going to their own doctor, automatically means
the probability of signi? cant disease is relatively high.
Clinical decision rules are widespread in Emergency
Medicine and help codify knowledge and explicitly de? ne pre -
test probability. However, unthinking application of such
decision - support tools by clinicians without an appreciation of
their ? aws and limitations results in bad decisions andor
over - investigation.
A history and examination taken by an experienced clinician
remains a very good predictor of pre - test probability of disease.
As can be seen opposite, a test applied in an inappropriate
population group, i.e. with a pre - test probability that is very
high or very low, will give misleading results. Tests are best
used in populations with an intermediate probability.
Using l ikelihood r atios in p ractice
It helps to think of the likelihood ratio as a multiplier that tells
you how much more or less likely a disease is, once you have the
result.
The particular advantage of likelihood ratios over other
measures of a test s performance is that, as shown opposite,they can easily be applied to individual patients, not just
populations.
A good test for ruling in a condition has a positive likelihood
ratio of more than 10, meaning that if the patient has a positive
test, it is 10 times more likely that they have the disease as a
result of the positive test.
For example, if a very speci? c test, e.g. Troponin I, is positive,we know that myocardial damage has occurred, because the
number of false positives (B) is very low. Similarly, if a very
sensitive test, e.g. CT scan for abdominal aortic aneurysm (AAA),comes back as negative, we know that it is very unlikely that the
patient has an AAA as the number of false negatives (C) will be
very low.
However, Emergency Departments use many tests that are
not 100% sensitive or speci? c, and therefore a more powerful, but
less intuitive, model is necessary to understand these tests: likeli-
hood ratios , which are calculated from the speci? city and
sensitivity.
Treatment o rders
Once the likely diagnosis has been made, a set of treatment orders
needs to be decided and documented. A good acronym for this is
DAVID, e.g., for an elderly patient with an open fracture of the tibia:
· D iet – nil by mouth
· A ctivities – elevate limb
· V ital signs monitoring – hourly limb observations
· I nvestigations – CXR, FBC, U + E
· D rugstreatment – immediate i.v. antibiotics
Test result
Actual patient status (truth)
Disease present Disease absent
Positive True positive (A) False positive (B)
Negative False negative (C) True negative (D)
Total no.
patients
With disorder (A + C) Without disorder
(B + D)
Sensitivity = A(A + C) and Speci? city = D(B + D ) 14 Adult Emergency Medicine at a Glance, 1st edition. ? Thomas Hughes and Jaycen Cruickshank. Published 2011 by Blackwell Publishing Ltd.
3 Shock and i ntravenous ? uids
Cannula (actual size) Flow rate (mlmin)
Shock: causes
No pressure
Pump failurepump blockage Distribution failure Fluid failure
Cardiac failure PE
Cardiac tamponade
Septic shock
Anaphylaxis
Spinal shock
Wrong way
Blood loss, e.g. GI bleed
Penetrating trauma
300 mmHg
Pink 20G
Green 18G
Grey 16G
Orange 14G
50 mlmin
100 mlmin
200 mlmin
350 mlmin
100 mlmin
200 mlmin
400 mlmin
700 mlmin
Above 14G the diameter of the fluid delivery tube
may be the limiting factor
Intravenous ? uid therapy is a common medical treatment, but
recently there has been a reassessment of the role of intravenous
· uids as some of the hazards have become better understood.
Intravenous a ccess
Poiseulle s Law governing ? uid ? ow through a tube (assuming
laminar ? ow):
Flow
L
∝ Pr
4
η
where P = pressure difference, r = radius, η = viscosity, L = length.
Therefore the ideal resuscitation ? uid should be non - viscous,driven by pressure through a short, wide cannula. For resuscita-
tion, or when giving blood (viscous), a 16 G or larger cannula is
preferred. The large veins in the antecubital fossa and the femoral
vein are good for resuscitation but are prone to infection and
uncomfortable for patients in the long term. A pressure bag
in? ated to 300 mmHg doubles ? uid ? ow.
Before cannulation the skin should be thoroughly cleaned using
chlorhexidine in alcohol. A cannula in the forearm is (relatively)
comfortable for the patient and less likely to become infected
compared to other sites. Shock and intravenous ? uids 15
Special c ases
· Central lines are very useful in very sick patients, patients with
poor access or patients whose ? uid balance is particularly dif? cult
to regulate. Their length means that they are not ideal for deliver-
ing resuscitation ? uids. Introducer sheaths offer a large bore
central access.
· Intraosseus needles have to be drilled into adult bone to give
· uid, but can be life - saving. Bone marrow aspirate may be used
for blood cross - matching.
· Venous cut - down involves cutting the skin to be able to cannulate
a vein under direct vision. The long saphenous vein 1 cm above
and anterior to the medial malleolus, or the basilic vein in the
antecubital fossa, are the most common sites for this.
Types of i ntravenous ? uids
Crystalloids
Normal saline, Hartmann s solution and Ringer s lactate are solu-
tions that match plasma osmolality. All can be used to resuscitate
patients, and despite vigorous debate, no one variety has proven
superior clinical outcomes.
Dextrose
50% dextrose is used to resuscitate hypoglycaemic patients. 10%
dextrose is used to maintain a patient s blood sugar and prevent
hypoglycaemia, and 5% is used to give ‘ free water to avoid over-
loading with sodium or chloride.
Colloids
Colloids contain large molecules that help retain ? uid within
the intravascular space, which improves blood pressure in the
short term. Unfortunately these molecules leak out of damaged
capillaries, which may cause resistant oedema in the brain and
lungs, which increases mortality in head - injured patients. Colloids
may be helpful in sepsis, but should only be used by senior doctors.
Blood
A full cross - match takes 30 minutes but type - speci? c blood should
be available within minutes. If blood is needed before the blood
type is known, Group O Rhesus negative blood is used.
Whole blood as donated is the best substitute in trauma, but has
a short shelf life (days). Separating blood cells into ‘ packed cells
extends storage time to 3 months, but deterioration may mean that
the cells are not fully functional for 24 hours. The citrate used to
stabilise blood binds calcium ions, which can cause problems in
massive transfusions ( > 50% blood volume).
Fresh frozen plasma (FFP) or synthetic clotting factors can be
used to correct clotting problems. Tranexamic acid, platelets and
FFP are given as part of massive transfusion protocol.
Temperature
Evolution has given humans enzymes that function best at 37 ° C
and pH ≈ 7.4. Blood clotting is impaired in a cold acidotic patient,e.g. trauma patient. Temperature < 34 ° C and pH < 7.20 reduce
clotting to 1% of normal. Laboratory measurements at 37 ° C will
not accurately re? ect the clinical picture. For this reason, clotting
factors are given early in trauma resuscitation. Cold ? uids (4 ° C)
may be given after cardiac resuscitation as part of an active cooling
strategy to preserve brain function.
Shock
Shock is de? ned as inadequate tissue perfusion, i.e. not meeting
the metabolic demands of tissue. Pulse and blood pressure are
bedside measures of tissue perfusion, but are insensitive. pH, P CO 2 ,lactate and mixed venous blood oxygen levels, measured from a
central venous pressure (CVP) line, are better indicators.
Types of s hock
The body pumps a limited amount of ? uid around a series of
closed loops. Problems occur when the ? uid disappears, the pump
fails or the ? uid goes to the wrong loops.
Blood f ailure
Blood loss may be controlled or uncontrolled, internal or external.
Severe dehydration may cause similar problems.
Pump f ailure
The heart may fail due to internal pump problems, e.g. myocardial
infarction or heart failure, which impair the ability to pump.
Alternatively the pump may fail because there is in? ow obstruc-
tion (cardiac tamponade, tension pneumothorax) or out? ow
obstruction (pulmonary embolus, aortic dissection).
Distribution f ailure
Blood may be distributed to the wrong organs. Inappropriate
vasodilation occurs in septic shock, anaphylaxis and spinal shock
(due to loss of sympathetic tone below the injury) diverting blood
away from vital organs.
Grades of s hock
Compensated shock BP → HR ↑
Young adults are able to compensate for loss of blood volume by
vasoconstriction and increased cardiac output, maintaining a good
BP and perfusion of vital organs.
Decompensated shock BP ↓ HR ↑
The body s compensation mechanisms are overwhelmed, and the
blood pressure falls rapidly.
Fluid r esuscitation
Traditional t eaching: ‘ Fill ‘ e m u p
· Good blood pressure = good outcome.
· Poor blood pressure = poor outcome.
· Therefore give ? uidblood to achieve good blood pressure.
Unfortunately this is an oversimpli? cation. Short - term poor
perfusion is well tolerated and if blood loss has not been
controlled:
· ↑ blood pressure = ↑ blood loss.
Increased blood loss is due to loss of vasospasm, dilution of clot-
ting factors and dislodgement of clot.
Current t eaching: ‘ m inimal v olume ? uid r esuscitation
If there is uncontrolled bleeding (e.g. penetrating trauma, ruptured
AAA), large - bore intravenous access is obtained. The minimum
volume of ? uid necessary to maintain cerebral perfusion or a
systolic BP of 60 – 80 mmHg is used ( ‘ permissive hypotension ). The
priority is urgent control of bleeding in the operating theatre.
Exception: if there is brain injury, the need to maintain cerebral
perfusion pressure overrides hypotensive resuscitation. 16 Adult Emergency Medicine at a Glance, 1st edition. ? Thomas Hughes and Jaycen Cruickshank. Published 2011 by Blackwell Publishing Ltd.
4 Imaging in the Emergency Department
The FAST scan is performed to look for free abdominal fluid – usually blood.
The fluid tends to pool in certain areas within the peritoneal cavity
Focussed Assessment using Sonography in Trauma – FAST Scan
Fluid in pericardial sac ± tamponade
Recto-vesical pouch
Recto-uterine pouch Hepato-renal pouch
Cardiac view
Free fluid in the pelvis (requires full bladder)
Pelvic view
Normal – no fluid between kidney and spleen
Spleno-renal view
Normal
Positive
Shows fluid (blood) between liver
and kidney
Hepato-renal view (Morison’s Pouch)Imaging in the Emergency Department 17
Imaging use in the Emergency Department has increased rapidly
over the past few years due to technical advances and increasing
pressure to move decision - making earlier in a patient s journey,and to prevent unnecessary hospital admissions. Ultrasound is
now a core skill for senior Emergency Department doctors and
new hospitals often have a CT scanner in the Emergency
Department.
Plain r adiography
Plain radiographs interpreted by the treating clinician are used for
the majority of Emergency Department imaging. The advent of
digital radiography has made real - time reporting by radiologists
easier.
X - rays are ionising radiation and cause damage to tissues
through which they pass. The energy released is proportional to
the density of the tissue. Abdominal or thoracolumbar radio-
graphs should not be performed in young people, especially
females, without a very good reason, as the gonads are very radio-
sensitive. In this book, X - ray doses are expressed in terms of chest
radiographs (CXR). One CXR is approximately 3 days of back-
ground radiation.
X - rays are not therapeutic. If the result will not change manage-
ment, radiographs should not be taken. Examples include uncom-
plicated rib fractures (when not worried about a pneumothorax),coccyx pain and stubbed toes other than the big toe. Soft tissues
are poorly shown by plain ? lms, making it an insensitive examina-
tion for joints that rely on these for stability, e.g. knee, shoulder.
Reading p lain r adiographs
1 Check the patient s name and the date of the ? lm, particularly
on digital radiography systems, which offer many opportunities
for confusion.
2 There should be two good views of limbs: anterior - posterior and
lateral.
3 If requesting imaging of more than one area, ask yourself if this
is necessary. If not urgent, it may be better to re - examine the patient
once they have had some analgesia, or obtain a senior opinion.
4 You will learn more from your radiology department if you
engage with them and ask their advice rather than expecting a
purely technical service.
5 Many Emergency Departments operate a system whereby the
radiographer can ? ag an abnormality on the radiograph. You
should not dismiss something that the radiographer has ? agged as
abnormal without obtaining a senior opinion.
Clinical u ltrasound
Clinical (bedside) ultrasound use has increased exponentially with
the availability of cheap robust ultrasound machines, and is now
a core skill for Emergency Department doctors. Ultrasound has
been described as the ‘ visual stethoscope and is revolutionising
the assessment and management of patients in the Emergency
Department.
Ultrasound was initially used in the Emergency Department in
the resuscitation room for:
· Detecting abdominal aortic aneurysms (AAA).
· Focused abdominal scanning in trauma (FAST) scans, searching
for blood in the peritoneal cavity.
· Central venous line placement.
However, ultrasound use is now expanding to include:
· Shock assessment: cardiac function, vascular ? lling, signs of
pulmonary embolus, together with the AAA and FAST scans.
· Basic echocardiography.
· Deep vein thrombosis (DVT) scanning.
· Early pregnancy scanning.
· Hepato - biliary scanning.
Disadvantages are that ultrasound is operator dependent,requires training and skill validation, and can divert attention
from more important problems.
Computed t omography s can
As resolution and availability have increased and acquisition
times have dropped, computed tomography (CT) has become
an increasingly useful tool for the Emergency Department. CT is
very good for bony injuries, and the trauma CT has proved to
be more sensitive and speci? c than clinical examination in
major trauma, but requires a very large radiation dose (1000
CXR).
Neck imaging in high - risk trauma is routinely done by CT (100
CXR), as plain ? lms are insuf? ciently sensitive at detecting signi? -
cant injury. Examples of high - risk injuries are a high - speed rollo-
ver road traf? c collision, and also the elderly patient who falls
forward, hitting their face ( ‘ fall on outstretched face ), who is at
high risk of odontoid peg fracture, and in whom interpretation of
plain radiographs is very dif? cult (see Chapter 11 ).
Modern CT scanners have enough resolution and speed to be
able to resolve cardiac anatomy including the coronary arteries,pulmonary emboli and aortic dissection (400 CXR). CT brain scan
(100 CXR) is an essential part of the assessment of stroke or the
unconscious patient. CT KUB (kidneys, ureters and bladder; 400
CXRs) is the imaging of choice in renal colic.
Magnetic r esonance s can
Magnetic resonance (MR) scanning is rarely used in the Emergency
Department apart from possible cauda equina syndrome (acute
central disc prolapse pressing on the cauda equina), giving bowel
and bladder symptoms. MR scanning can be used to avoid the
large radiation dose incurred by CT, e.g. investigating renal colic
in young women.
Joints in which stability and function are mainly due to soft
tissues, i.e. ligaments and cartilage such as the knee and shoulder,are well imaged by MR scanning, but it is generally dif? cult to
access these directly from the Emergency Department.
Interventional i maging
Interventional imaging has an increasing role for a limited number
of severe conditions. Interventional imaging is generally offered
in larger hospitals, and together with trauma care, is one of the
main drivers for centralisation of acute services into large
hospitals.
· Primary percutaneous cardiac intervention with stenting has
become the treatment of choice for patients with myocardial
infarction.
· Endovascular treatments for patients with AAAs and aortic dis-
section are increasingly used. Neurosurgical bleeding from aneu-
rysms is treatable by coils, as is otherwise uncontrollable bleeding
in the pelvis, e.g. from pelvic fractures. 18 Adult Emergency Medicine at a Glance, 1st edition. ? Thomas Hughes and Jaycen Cruickshank. Published 2011 by Blackwell Publishing Ltd.
5 Analgesia
Intravenous regional anaesthesia
Non-pharmacological analgesia Analgesic ladder
Splint Plaster
Reduce dislocation
Sling, elevation,patient
positioning
Raise arm, inflate cuffs
↑300 mmHg
1 Inject prilocaine 2 Manipulate fracture,plaster, X-ray
3 Minimum 20 minutes later 4
↓0 mmHg
NSAID
Strong
Weak
Opiates
Ketorolac Morphine
Indometacin
Tramadol
Diclofenac
Ibuprofen Codeine
Paracetamol (acetaminophen)
Patients often arrive at the Emergency Department in pain, and
painkillers are often used before a de? nitive diagnosis is made.
This is humane, and enables a thorough examination to be per-
formed: there is no reason to withhold analgesia.
Patients are asked to rate the pain out of 10, with 0 being no
pain, and 10 being the worst pain they can imagine. This procedure
is repeated to gauge the effectiveness of the treatment and ensure
the pain is controlled.
In general, a patient s reported pain is taken at face value: ‘ pain
is what the patient feels and is treated as such. Patients seeking
opiates may fake pain, but this is rare.
Non - p harmacological a nalgesia
Splinting of fractures immobilises the bones, reducing pain. A
patient s anxiety and pain makes them tense, which may make
pain worse: a calm, supportive atmosphere and excellent nursing
care help to keep the patient relaxed.
Nitrous o xide
Nitrous oxide (N 2 O) combined with oxygen in a 1 : 1 mix in cylin-
ders (Entonox ? ) is often used, particularly out of hospital. It is a
short - term analgesic, effective only while the patient is breathing Analgesia 19
the gas, as it is rapidly cleared from the body. This ‘ laughing gas
is generally very safe, but should not be used in patients with a
possible pneumothorax.
Paracetamol ( a cetaminophen) and
c ompound a nalgesics
Paracetamol (acetaminophen) is effective and safe and can be
given orally, rectally or intravenously. Compound analgesics
consist of paracetamol combined with another analgesic, usually
low - dose codeine. They come in different strengths, the weaker of
which are sold without prescription. They are useful analgesics for
patients to be able to take home on discharge, but prescribing the
constituent drugs separately may allow more ? exibility.
Moderate o piates
· Codeine is a common component of compound analgesics, and
is effective but tends to cause constipation. Oxycodone and dihy-
drocodeine are more powerful variants of codeine, but offer little
extra bene? t, and have high abuse potential.
· Tramadol may be more effective than codeine. It has less abuse
potential than other drugs of comparable potency but should be
used with caution in the elderly.
Major o piates: m orphine, f entanyl,p ethidine ( m eperidine)
Opiates induce a feeling of well - being: patients, while still aware
of the pain, are not distressed by it. Young patients with major
fractures may require large doses of morphine, as will opiate
addicts who need analgesia. Intravenous opiates are used because
intramuscular absorption is unreliable and the intravenous route
enables analgesia to be titrated to response.
· Intravenous morphine is the gold standard of Emergency
Department analgesia. It is safe, predictable and effective.
Morphine is not as lipid soluble as other opiates, so does not give
a signi? cant ‘ high . Morphine often causes mild histamine release
that should not be confused with an allergic reaction. The duration
of action of morphine is approximately 3 hours.
· Fentanyl is a short - acting synthetic opiate that is particularly
useful when performing short procedures, as it is cleared from the
body within 30 minutes.
· Pethidine (meperidine) is quite lipid soluble and therefore sought
after by opiate addicts as it crosses the blood – brain barrier, giving
a ‘ high . It offers no bene? ts over morphine and should not be used
unless a patient has a de? nite allergy to morphine and there are
no other alternatives.
Non - s teroidal a nti - i n? ammatory d rugs
Injectable non - steroidal anti - in? ammatory drugs (NSAIDs), e.g.
ketorolac , are very effective in an Emergency Department setting.
They are particularly useful in patients with broken bones, colicky
pain (e.g. ureteric colic) and abdominal pain, but should be
avoided in elderly patients or those with active bleeding. An
equally effective alternative is a suppository (e.g. indometacin,diclofenac), which lasts for 16 hours.
Oral NSAIDs are useful as they can also be given to patients on
discharge. Ibuprofen is the least powerful, but has a relatively
benign side - effect pro? le.
Diclofenac and indometacin are more powerful NSAIDs but at
a cost of increased risk of side - effects.
Local a naesthesia and n erve b locks
· Lidocaine 1% is the local anaesthetic (LA) most often used for
wound management and is effective for 20 – 30 minutes without
adrenaline, or for 40 – 60 minutes with adrenaline.
· Adrenaline mixed with lidocaine increases length of action and
causes vasoconstriction giving a ‘ dry wound that is much easier
to assess, clean and close. Fear about using local anaesthetics with
adrenaline in digits was related to high concentrations (1 : 10 000);
less than 1 : 100 000 adrenaline is safe.
· Bupivicaine 0.25% is a long - acting local anaesthetic, lasting for
6 – 8 hours. Bupivicaine is highly protein bound: adrenaline does
not increase duration of action.
A safe maximum dose of lidocaine for wound in? ltration is 3 mg
kg, but with adrenaline is 6 mgkg. For bupivicaine the maximum
dose is 2 mgkg. Local anaesthetic toxicity ? rst causes perioral
parasthesia, and then ? ts and arrhythmias, and is treated by lipid
infusion.
Nerve blocks can offer very effective analgesia, e.g. digital and
femoral nerve blocks. Bupivicaine and lidocaine can be mixed to
provide a combination of rapid onset and long duration of action.
Local anaesthetic can also be injected into joints, e.g. for shoulder
dislocation.
A haematoma block can give good anaesthesia in minor frac-
tures e.g. Colle s fractures (Chapter 15 ). The skin is carefully
cleaned with alcohol and chlorhexidine and then up to 10 mL of
local anaesthetic is injected into the fracture haematoma. After
about 10 minutes reduction can be performed.
Intravenous r egional a naesthesia
(Bier s b lock)
Two intravenous cannulae are sited, one in the affected limb. A
double cuff is placed on the affected limb (usually the arm), which
is then lifted to exsanguinate it. The cuff is then in? ated well above
the systolic BP and local anaesthetic, e.g. prilocaine, injected.
Bupivicaine should never be used for intravenous regional
anaesthesia.
After waiting 5 minutes for the local anaesthetic to have maximal
effect, the operation, e.g. fracture reduction, is performed. The cuff
must not be de? ated until at least 20 minutes have elapsed from
injection of the local anaesthetic to avoid a bolus of undiluted local
anaesthetic perfusing the heart, potentially causing asystole. 20 Adult Emergency Medicine at a Glance, 1st edition. ? Thomas Hughes and Jaycen Cruickshank. Published 2011 by Blackwell Publishing Ltd.
6 Airway m anagement and s edation
Oropharyngeal airway
Measure from nose to tragus Measure from teeth to angle of jaw
Nasopharyngeal airway
Endotracheal tube in situ
Head tilt
Jaw thrust
Laryngoscope
Cricothyroid membrane
Endotracheal tube
View with a laryngoscope
Vocal cords
Epiglottis
Tongue
Needle cricothyroidotomy Surgical airway
Laryngoscope
Size 6 cuffed
endotracheal tube
Once the incision has been made NEVER let go
Check CO2
CXR
Oxygen tube
Three way tap with
extension tube
Cannula
Macintosh blade,size 3
8.0
7.0
Thyroid cartilage Cricothyroid
membrane
Cricoid
cartilage
Airway management in the Emergency Department is more chal-
lenging than in the operating room as patients presenting to the
Emergency Department must be assumed to be non - fasted, may
be physiologically unstable, and may have head, neck or facial
injuries.
Oxygenation and v entilation
Oxygenation is ensuring that the body has enough O 2 ; ventilation
is ensuring that there is suf? cient air? ow to remove CO 2 . Oxygen
consumption is markedly increased in the acutely unwell patient,and giving high concentrations of oxygen supports the metabolic
demands of the body in acute illness. However, high levels of
oxygen may paradoxically make some ischaemic injury worse, e.g.,brain heart due to vasoconstriction. A normal ‘ Hudson O 2 mask
can give inspired oxygen (FiO 2 ) concentrations of up to 60%. They
should not be used with O 2 < 4 Lmin to prevent CO 2 build - up. A
mask with a reservoir bag or a self - in? ating bag - valve - mask can
increase FiO 2 to about 90% with high ? ow ( > 10 Lmin O 2 ). A Venturi mask gives accurate low FiO 2 e.g. 28%. Nasal prongs give
a variable amount of O 2 approx 25 – 30% but should only be used
with low ? ow rates (2 Lmin O 2 ).
Ventilatory f ailure
Under normal circumstances, an increased level of CO 2 is the main
driver to breathe. Patients with chronic lung disease, usually
COPD, become immune to this drive. For these ‘ blue bloater
patients, a low blood O 2 level drives breathing: their CO 2 level will
be high.
If high FiO 2 is given to these patients, it reduces their respiratory
drive, increasing their CO 2 levels further, making them sleepy,which further decreases their drive to breathe, etc. An oxygen satu-
ration target of 91% in these patients balances the need for tissue
oxygenation against that for ventilation.
· O 2 should be prescribed, with a target saturation
· A patient with O 2 sat > 96% probably does not need extra O2 unless
high metabolic need, e.g., sepsis, trauma
· If FiO 2 has given a very high CO 2 level, reduce FiO 2 slowly
· If in doubt, give O 2 and obtain a senior review
Suction
A Yankauer suction catheter is used to suction blood, vomit or
secretions in the oropharynx. To avoid causing the patient to
vomit, do not suction the oropharynx if the patient is conscious,and ‘ only suck where you can see .
Airway s upport
The jaw thrust, head tilt, oropharyngeal and nasopharangeal airways
are illustrated opposite . The oropharyngeal airway is sized as the
distance between the patient s teeth and the angle of the mandible.
The nasopharyngeal airway should be the same length as the distance
between the tip of the nose and the tragus of the ear.
Laryngeal m ask a irway
Emergency Department patients are not fasted and the laryngeal
mask airway (LMA) does not prevent stomach contents being
aspirated, nor can high ventilation pressures be achieved, as might
be necessary in asthmatic patients. For these reasons the LMA is
not a ‘ de? nitive airway and is not normally used in the Emergency
Department.
Endotracheal t ube
The most common means to provide a de? nitive airway, the endotra-
cheal tube (ETT), is a plastic tube that is inserted through the mouth
(or rarely the nose) into the trachea. There is a cuff that is in? ated to
seal against the tracheal mucosa, and a radio - opaque line to indicate
position on X - ray. The ETT should be secured, e.g. with tape, and
the position checked by CO 2 monitoring and a chest X - ray.
Endotracheal tubes are sized by their internal diameter: 7.0 mm
for an adult female, 8.0 mm for a male. There are markings indicat-
ing distance from the tip: this is to avoid the tube being pushed
WARNING
Airway management and sedation 21
too far, e.g. down the right main bronchus, which is larger and
straighter than the left.
The decision that the patient needs intubation is the responsibil-
ity of the doctor managing the airway. Factors indicating need for
intubation include:
· Airway instability: bleeding into airways, airway burns.
· Coma: Glasgow Coma Scale (GCS) < 9, deteriorating level of
consciousness, loss of protective laryngeal re? exes.
· Inadequate oxygenation: despite high inspired O 2 (FiO 2 ).
· Inadequate ventilation: patient tireddrowsy.
· Therapeutic reasons: control seizures, hypothermia.
· Pragmatic reasons: combative patient, need for transport.
A laryngoscope is needed to insert the ETT. In some countries,straight (Miller) blades are used; in others, curved (Macintosh)
blades. These have a light to enable sight of the larynx.
McGill s forceps have a ‘ kink in them to avoid the operator s
hands obstructing the ? eld of vision. They are useful for removing
loose items in the oropharynx, and manipulating the ETT.
Surgical a irway
Rarely, a situation occurs when it is not possible to intubate or
ventilate a patient. In this situation, there are two options:
· A needle cricothyroidotomy will provide short - term oxygenation,but is not a de? nitive airway, and CO 2 levels will build up.
· A surgical airway through the cricoid membrane using a 6.0 mm
cuffed ETT provides a de? nitive airway.
Procedural s edation
Procedural sedation is often performed in the Emergency
Department to allow relocation of dislocations or for short painful
procedures. The person performing the sedation needs appropriate
skills and experience to manage any potential situation, including
the need for intubation.
The procedure should be carried out in a resuscitation bay with
full monitoring, oxygen and suction equipment. Two doctors
should be present at all times to ensure that the doctor administer-
ing the sedation has their full attention on the patient s airway.
The patient should be fasted for at least 4 hours, should give
formal consent, and the doctor should stay with ......
Medicine at a Glance
Thomas Hughes
Bt.MSC, MBA, MRCP, FRCS, FCEM
Consultant Emergency Physician
John Radcliffe Hospital, Oxford
Honorary Senior Lecturer in Emergency Medicine
University of Oxford
Jaycen Cruickshank
MCR, FACEM
Director of Emergency Medicine
Ballarat Health Services, Victoria, Australia
Senior Lecturer in Emergency Medicine
University of Melbourne, Victoria, Australia
A John Wiley Sons, Inc., PublicationThis edition ? rst published 2011, ? 2011 by Thomas Hughes and Jaycen Cruickshank
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Library of Congress Cataloging-in-Publication Data
Hughes, Thomas, MSc.
Adult emergency medicine at a glance Thomas Hughes, Jaycen Cruickshank.
p. ; cm.
Includes index.
ISBN 978-1-4051-8901-9
1. Emergency medicine--Handbooks, manuals, etc. I. Cruickshank, Jaycen. II. Title.
[DNLM: 1. Emergency Medicine--methods--Handbooks. 2. Adult. WB 105 H894a 2011]
RC86.8.H84 2011
616.02'5--dc22
2010024551
ISBN: 9781405189019
A catalogue record for this book is available from the British Library.
Set in 9 on 11.5 pt Times by Toppan Best-set Premedia Limited
1 2011Contents 5
Contents
Preface and acknowledgements 7
List of abbreviations 8
1 Life in the Emergency Department 10
2 Diagnosis 12
3 Shock and intravenous ? uids 14
4 Imaging in the Emergency Department 16
5 Analgesia 18
6 Airway management and sedation 20
7 Blood gas analysis 22
8 Trauma: primary survey 24
9 Trauma: secondary survey 26
10 Major head and neck injury 28
11 Minor head and neck injury 30
12 Wounds 32
13 Burns 34
14 Hand injuries 36
15 Wrist and forearm injuries 38
16 Shoulder and elbow injuries 40
17 Back pain, hip and knee injuries 42
18 Tibia, ankle and foot injuries 44
19 Abdominal pain 46
20 Urology problems 48
21 Ear, nose, throat and dental problems 50
22 Eye problems 52
23 Obstetrics and gynaecology problems 54
24 Toxicology: general principles 56
25 Toxicology: speci? c poisons 58
26 Psychiatry: self-harm and capacity 60
27 Psychiatry: the disturbed patient 62
28 Observational medicine 64
29 Loss of function and independence 66
30 Syncope, collapse and falls 68
31 Slow heart rate 70
32 Fast heart rate 72
33 Cardiac arrest 74
34 Chest pain: cardiovascular 76
35 Chest pain: non-cardiovascular 78
36 Shortness of breath 80
37 Anaphylaxis 82
38 Sepsis 84
39 Endocrine emergencies 86
40 Gastroenterology 88
41 Headache 90
42 Stroke and transient ischaemic attack 92
43 Seizures 94
44 Hypothermia and hyperthermia 96
45 Pre-hospital medicine 98
46 Major incident 100
47 Chemical, biological, radiation, nuclear and explosive
incidents 102
Case studies: questions 104
Case studies: answers 107
Index 113Preface and Acknowledgements 7
Preface
Emergency Medicine has undergone a quiet revolution over the
past twenty years due to a variety of factors that have changed the
way medicine is practiced.
· Increasing demand and expectations of medical care.
· Reduction of junior doctors hours.
· An ageing population.
· Fragmentation of out of hours care.
· Reduced hospital bed - stay.
· Sub - specialisation of inpatient medical and surgical practice.
· Litigation.
These factors have pushed expert decision - making towards
the front door of the hospital so that the correct diagnosis and
treatment start as soon as possible in the patient s journey. As
other specialties have moved away from the acute assessment and
treatment of patients, Emergency Medicine has expanded to ? ll
the vacuum left, and in doing so has increased its realm of practice
substantially.
Emergency Medicine is exciting and confronting, intimidating
and liberating – it is the chance to exercise and hone your diag-
nostic and practical skills in a well - supervised environment.
Clinical staff who work in the ED have all been through the
inevitable feelings of fear, uncertainty and doubt that come
with the territory, and want you to experience the enjoyment
and satisfaction of working in an area of medicine that is never
boring.
When trainees start Emergency Medicine, it is often the ? rst
time they have seen patients before any other staff. To use a tra-
ditional analogy, they have seen plenty of needles, and may be very
good at recognising them, but now they are faced with haystacks,in which may be hidden a variety of sharp shiny objects.
Medical textbooks usually describe topics by anatomy or pathol-
ogy (needles), e.g. heart failure, which tends to assume the diag-
nostic process. In this book we have tried to organise topics by
presentation (haystacks), e.g. ‘ short of breath , and have tried to
articulate the key features that help us ? nd the needles.
We are both great fans of the ‘ At a Glance series, and have
enjoyed the challenge of combining the breadth of practice of adult
Emergency Medicine with the concise nature of the ‘ At a Glance
format. We hope you enjoy this book and ? nd it useful as you
explore this most dynamic area of medicine.
Acknowledgements
We would like to thank Karen Moore and Laura Murphy at
Wiley - Blackwell for their support and advice (and let s face it,patience) during the elephantine gestation of the book. Also Helen
Harvey for understanding deadlines, and Jane Fallows for doing
such a great job with the illustrations. We thank the students of
Oxford and Melbourne Universities, whose inquiring minds keep
us on our toes, and whose questions stimulated us to think of new
ways to present the information we have used here. We are both
lucky enough to have worked with an exceptional colleague, Philip
Catterson, whose teamwork, leadership and hard work have
helped us to achieve success in our jobs, and from whom we have
learned the few interpersonal skills we have.
In addition TH would like to thank: Professor Christopher
Bulstrode who has been an inspiration and mentor and without
whose support the book would not have happened. My family,and particularly my wife Marina for her support. My work col-
leagues for continuing to tolerate me most of the time, and Jackie
and Tracey, the ED secretaries who keep me in order. Nic Weir,Rob Janas and David Bowden for their help in sourcing images
and the ? nal preparation.
JC would like to thank: All the people who have taught me
along the way, particularly Trevor Jackson and Steven Pincus. My
parents Ron and Christine for making everything possible, and my
wife Kerry and sons Jesse and Flynn for making everything
worthwhile.
Thomas Hughes
Jaycen Cruickshank 8 List of abbreviations
List of abbreviations
ED Emergency Department
EDTA ethylene diamine tetraacetate
ELISA enzyme - linked immunosorbent assay
ENT ear, nose and throat
EPL extensor pollicis longus
ESR erythrocyte sedimentation rate
ETT endotracheal tube
FAST acronym for focused abdominal sonography in trauma;
also face, arm, speech, time to call ambulance
FB foreign body
FBCFBE full blood countexamination
FiO 2 fraction of inspired of oxygen (as %)
FFP fresh frozen plasma
FOOSH fall onto an outstretched hand
GA general anaesthetic
GA β HS group A β - haemolytic Streptococcus
GCS Glasgow Coma ScaleScore
GI gastrointestinal
GP general practitioner
H1N1 swine ? u virus
H5N1 avian ? u virus
HbA 1c glycated (glycosylated) haemoglobin
HCO3--
bicarbonate ion
hCG human chorionic gonadotrophin
HDU high dependency unit
HHS hyperosmolar hyperglycaemic state
HIV human immunode? ciency virus
HOCM hypertrophic obstructive cardiomyopathy
HONK hyperosmolar non - ketotic acidosis
HR heart rate
HVZ herpes varicella zoster
IBS irritable bowel syndrome
ICP intracranial pressure
ICU intensive care unit
IgE immunoglobulin E
IVDU intravenous drug use
IVF in vitro fertilisation
IVRA intravenous regional anaesthesia
IVU intravenous urogram
JVP jugular venous pressure
KUB kidneys, ureters and bladder
LA local anaesthetic
LCL lateral collateral ligament of knee
LFT liver function test
LMP last menstrual period
LNMP last normal menstrual period
LOC loss of consciousness
LP lumbar puncture
LR likelihood ratio
LRTI lower respiratory tract infection
MAOI monoamine oxidase inhibitor
MAP mean arterial pressure
MCL medial collateral ligament of knee
MCPJ metacarpophalangeal joint
MDI metered dose inhaler
AAA abdominal aortic aneurysm
ABC airway, breathing, circulation
ABCD
2
acronym to assess stroke risk in a patient with TIA
ABCDE airway, breathing, circulation, disability, exposure
ABG arterial blood gases
ACE angiotensin - converting enzyme
ACh acetylcholine
ACJ acromioclavicular joint
ACL anterior cruciate ligament of knee
ACS acute coronary syndrome
ACTH adrenocorticotrophic hormone
AD aortic dissection
AF atrial ? brillation
AIDS acquired immunode? ciency syndrome
AMT4 four-point abbreviated mental test score
AP antero - posterior
APL abductor pollicis longus
AV arteriovenous; also atrioventricular
AVN atrioventricular node
AXR abdominal X - ray
BDZ benzodiazepine
BP blood pressure
bpm beats per minute
CAGE acronym for alcohol screening questions
CAP community - acquired pneumonia
cAMP cyclic adenonsine monophosphate
CBRNE chemical, biological, radiological, nuclear,explosive
CK creatine kinase
CNS central nervous system
CO carbon monoxide
COHb carboxyhaemoglobin
COPD chronic obstructive pulmonary disease
CPAP continuous positive airway pressure
CPP cerebral perfusion pressure
CPR cardiopulmonary resuscitation
CRAO central retinal artery occlusion
CRP C - reactive protein
CRVO central retinal vein occlusion
CT computed tomography
CTPA CT pulmonary angiography
CURB - 65 confusion, urea, respiratory rate, blood pressure,age over 65 (acronym for pneumonia severity
factors)
CVP central venous pressure
CXR chest X - ray; also unit for X - ray dose, 1 CXR ≈ 3
days background radiation
DIPJ distal interphalangeal joint
DKA diabetic ketoacidosis
DM diabetes mellitus
DSH deliberate self - harm
DUMBELS diarrhoea, urination, miosis, bronchorrhoeabron-
chospasm, emesis, lacrimation, salivation (acronym
for clinical effects of organophosphate poisoning)
DVT deep vein thrombosis List of abbreviations 9
MI myocardial infarction
MR magnetic resonance
N 2 O nitrous oxide
NAC N - acetylcysteine
NICE National Institute for Health and Clinical Excellence
NIV non - invasive ventilation
NNT number needed to treat
NNH number needed to harm
NoF fractured neck of femur
NSAID non - steroidal anti - in? ammatory drug
NSTEMI non - ST segment elevation myocardial infarction
OD overdose
OP organophosphate
OPG oral pantomogram
ORIF open reduction and internal ? xation
PA postero - anterior
PCL posterior cruciate ligament of knee
PE pulmonary embolism
PEA pulseless electrical activity
PEF peak expiratory ? ow
PEFR peak expiratory ? ow rate
PID pelvic in? ammatory disease
PPCI primary percutaneous coronary intervention
PPI proton pump inhibitor
PPM permanent pacemaker
PR per rectum
PT prothrombin time
PV per vaginam
RA regional anaesthesia
RBBB right bundle branch block
RoSC return of spontaneous circulation
SAH subarachnoid haemorrhage
SAN sinoatrial node
SARS severe acute respiratory syndrome
SDH subdural haematoma
SoB short(ness) of breath
SOCRATES acronym for pain history
SOL space - occupying lesion
SSRI selective serotonin reuptake inhibitor
STD sexually transmitted disease
STEMI ST segment elevation myocardial infarction
STI sexually transmitted infection
SVT supraventricular tachycardia
TBSA total body surface area
TCA tricyclic antidepressant
TFT thyroid function test
TIA transient ischaemic attack
TIMI thrombolysis in myocardial infarction
TMT tarsometatarsal
tPA tissue plasminogen activator
U + E urea and electrolytes
UA unstable angina
URTI upper respiratory tract infection
UTI urinary tract infection
VBG venous blood gases
VF ventricular ? brillation
VQ ventilationperfusion
VT ventricular tachycardia
VVS vasovagal syncope
WCC white cell count 10 Adult Emergency Medicine at a Glance, 1st edition. ? Thomas Hughes and Jaycen Cruickshank. Published 2011 by Blackwell Publishing Ltd.
1 Life in the Emergency Department
Patients with acute health needs
GP
Triage
1
Resus
2
High
acuity
– time
critical
3
High
acuity
– not time
critical
4
Low
acuity
5
Non-urgent
Community
ED
Discharged patients
Short stay
wards
Community
ED
Hospital
Cough
Inpatient
units
This chapter describes the way the Emergency Department oper-
ates, and some of the unwritten rules. The prevalence of Emergency
Department - based drama generates plenty of misconceptions
about what occurs in the Emergency Department. For instance,it is generally inadvisable to say ‘ stat at the end of one s
sentences, and neither of the authors has been mistaken for George
Clooney!
What h appens w hen a p atient a rrives
at the Emergency Department?
Alert p hone
Also known as the ‘ red phone or sometimes ‘ the Bat - phone , this
is the dedicated phone line that the ambulance service uses to pre -
warn the Emergency Department of incoming patients likely to
need resuscitation.
Triage
The concept of triage comes from military medicine – doing the
most good for the most people. This ensures the most effective use
of limited resources, and that the most unwell patients are seen
· rst.
Nurses rather than doctors are usually used to perform the
triage because doctors tend to start treating patients. Systems
of rapid assessment and early treatment by senior medical staff
can be effective, but risk diverting attention from the most ill
patients. Life in the Emergency Department 11
Reception r egistration
The reception staff are essential to the function of the Emergency
Department: they register patients on the hospital computer
system, source old notes and keep an eye on the waiting room.
They have to deal with dif? cult and demanding patients, and are
good at spotting the sick or deteriorating patient in the waiting
room.
Waiting r oom
Adult and paediatric patients should have separate waiting rooms,and some sort of entertainment is a good idea. Aggression and
dissatisfaction in waiting patients has been largely eliminated in
the UK by the 4 - hour standard of care: all patients must be
seen and discharged from the Emergency Department within 4
hours.
Treatment a reas in the
Emergency Department
Resuscitation b ays
Resuscitation bays are used for critically ill and unstable patients
with potentially life - threatening illness. They have advanced moni-
toring facilities, and plenty of space around the patient for clinical
staff to perform procedures. X - rays can be performed within this
area.
High a cuity a rea
This is the area where patients who are unwell or injured, but who
do not need a resuscitation bay, are managed. Medical conditions
and elderly patients with falls are common presentations in this
area.
Low a cuity a rea
The ‘ walking wounded – patients with non - life - threatening
wounds and limb injuries – are seen here. Patients with minor
illness are discouraged from coming to the Emergency Department,but continue to do so for a variety of reasons.
There is a common misconception that patients in this area are
similar to general practice or family medicine patients. Numerous
studies have found that there is an admission rate of about 5% and
an appreciable mortality in low acuity patients, whereas only
about 1% of GP consultations result in immediate hospital
admission.
Other a reas
Imaging
Imaging, such as X - rays and ultrasound, are integral to Emergency
Department function. Larger Emergency Departments have their
own CT scanner.
Relatives r oom
When dealing with the relatives of a critically ill patient and break-
ing bad news, doctors and relatives need a quiet area where infor-
mation is communicated and digested. This room needs to be close
to the resuscitation area.
Observationshort stay w ard
This is a ward area close to the Emergency Department, run by
Emergency Department staff. This unit treats patients who would
otherwise need hospital admission for a short time, to enable them
to be fully stabilised and assessed. The function of these units is
described in Chapter 28 .
Hospital in the h ome
Some hospitals run a ‘ hospital in the home programme for
patients who do not need to be in hospital but who need
certain therapy, e.g. intravenous antibiotics, anticoagulation. The
Emergency Department is the natural interface between home
and hospital.
Culture of the Emergency Department
There is a much ? atter (less hierarchical) organisational structure in
the Emergency Department than most other areas in the hospital.
This occurs because all levels of medical, nursing and other staff
work together all the time, and the department cannot function
without their cooperation. Ensuring good teamwork requires good
leadership, an atmosphere of mutual respect and a bit of patience
and understanding.
The resulting atmosphere can be one of the most enjoyable and
satisfying places to work in a hospital. A feature of this less hier-
archical culture that surprises junior doctors is that nurses will
question their decisions; this is a sign of a healthy culture in which
errors are less likely to occur, and is actively encouraged.
Emergency Department r ules
Being a doctor in the Emergency Department is different from
elsewhere in the hospital. There is nowhere to hide and, for
the ? rst time in most medical careers, you are responsible for
making the decisions. On the positive side, there are plenty
of people around to help you, who have all been through the
same process.
Some basic advice:
· Write legible, timed, dated notes.
· Show respect for other professional groups and be prepared to
learn from them.
· Do not be late for your shifts; do not call in sick less than 6 hours
before a shift.
· Patients who re - present are high risk and need senior review.
· Take your breaks. You need them.
· Keep calm.
· If in doubt, ask.
· Do not pick up so many patients that you cannot keep track of
them.
· Do not avoid work or avoid seeing dif? cult patients. We do notice.
· The nurse in charge is usually right.
With so many people working closely together in a stressful
atmosphere, it is inevitable that con? icts will occur. Do not
let them fester; some ground rules for resolving such con? icts
are:
· Resolve it now.
· Do it in private.
· Do it face to face.
· Focus on facts.
· Criticise action, not person.
· Agree why it is important.
· Agree on a remedy.
· Finish on a positive.
12 Adult Emergency Medicine at a Glance, 1st edition. ? Thomas Hughes and Jaycen Cruickshank. Published 2011 by Blackwell Publishing Ltd.
2 Diagnosis
10
1
0.1
ODDS 1:20
A type of d-dimer test has a specificity of 50% and a sensitivity of 95%:
this allows you to calculate the likelihood ratios to rule a condition in (+LR)
or out (–LR)
A woman presents with a history suggestive of DVT, and her
probability of having a DVT is low; about 5%, according to the
Wells score (see Ch. 35).
The d-dimer test comes back negative.
What is the chance she has a DVT?
Probability 5%
0.1:20 = 1:200
0.5%
0.1
1 – Sn 1 – 0.95
1 – Sp
= 1.9 Sn +LR =
1 – 0.5
0.95 = = 0.1 –LR =
Sn 0.5
=
10
1 x
x
=
x
x
=
x
x
=
0.1
ODDS 1:20
If the d-dimer test had come back positive, what is the chance she
has a DVT?
Probability 5%
1.9:20 = 1:10
10%
1.9
The result (0.5%) is the risk of missing a DVT in this patient.
Bearing in mind that no test is perfect, a result below 1% is
generally taken as an acceptable level of risk
10
1
0.1
x
x
=
10
1
0.1
ODDS 1:1
Probability 50%
0.1:1 = 1:10
10%
0.1 5 ODDS 1:100
Probability 1%
5:100 = 1:20
5%
Despite the positive d-dimer, this patient only has a 10% chance
of having a DVT
This shows that this d-dimer test is useful for excluding a DVT in a low-risk population, but that a positive test does not mean a DVT is present
A patient who has been immobile following a recent operation
for cancer has pain in his lower leg. You assess his risk of
DVT as about 50%, but the test comes back negative.
What is the probability he has a DVT?
Despite a negative d-dimer, he still has a 10% chance of having
a DVT. D-dimer cannot ‘rule out’ DVT in a high risk patient
A young man with mild suprapubic pain, whom you estimate has
a 1% chance of having a UTI, has a positive urine leucocyte test
(+ LR = 5) on dipstick testing. What is the chance he has a UTI?
Despite a positive test with a fairly high + LR he still only has a 5%
chance of having a UTI because the condition was so unlikely
in the first place
The Emergency Department is a diagnosis machine, taking
people with a wide variety of symptoms, labelling them with
a diagnosis, treating and then discharging them whenever
safe to do so. There are plenty of opportunities for this process
to go wrong, and it is important to understand how this can
occur.
‘ If you listen carefully to the patient, they will tell you the
diagnosis . W Osler
Despite a couple of thousand years of medical education, we are
still not really sure how the diagnostic process works.
· Some people work forward from history, examination and a
shortlist of differential diagnoses.
· Others work backwards from a list of likely diagnoses to weight
these according to symptoms and examination.
· Experts make their diagnoses by pattern recognition.
It may be that expertise occurs with the development of cogni-
tive ? exibility to use multiple diagnostic strategies to integrate and
test the result. Using t ests
In the past, the Emergency Department used a few simple tests
to inform decision - making. An X - ray of an injured limb has a
binary outcome: broken not broken. As technology and the scope
of Emergency Medicine has increased, the tests have become more
numerous and less black and white, and there is a need to ration-
alise and manage the uncertainty generated.
The most common way of describing a test s performance is to
use sensitivity and speci? city .
Confusion m atrix
Diagnosis 13
SpIN – a very Speci? c test rules a condition IN .
SnOUT – a very Sensitive test rules a condition OUT .
Bayes theorem
The chance of something being true or false depends not only
on the quality of the test that one is performing, but impor-
tantly , how likely the event is in the ? rst place.
Thomas Bayes, an eighteenth - century English priest, deduced
the principles that underpin the way we use tests in medicine:
pre - t est p robability × likelihood r atio = post - t est
p robability
To calculate this, we use odds (like horse racing odds) rather
than probability, but the two are obviously very closely related.
The likelihood ratio is calculated from the test s sensitivity
(Sn) and speci? city (Sp) and is a much better measure of a test s
clinical usefulness in ruling a disease in (positive LR) or out
(negative LR).
+=
·
·=
·
LR Sn
Sp
LR Sn
Sp 1
1
How d o I d e? ne p re - t est p robability?
The triage process uses an expert nurse to assess clinical status
and is an assessment of the probability of serious illness. The
fact that a patient has arrived at the Emergency Department at
all, rather than going to their own doctor, automatically means
the probability of signi? cant disease is relatively high.
Clinical decision rules are widespread in Emergency
Medicine and help codify knowledge and explicitly de? ne pre -
test probability. However, unthinking application of such
decision - support tools by clinicians without an appreciation of
their ? aws and limitations results in bad decisions andor
over - investigation.
A history and examination taken by an experienced clinician
remains a very good predictor of pre - test probability of disease.
As can be seen opposite, a test applied in an inappropriate
population group, i.e. with a pre - test probability that is very
high or very low, will give misleading results. Tests are best
used in populations with an intermediate probability.
Using l ikelihood r atios in p ractice
It helps to think of the likelihood ratio as a multiplier that tells
you how much more or less likely a disease is, once you have the
result.
The particular advantage of likelihood ratios over other
measures of a test s performance is that, as shown opposite,they can easily be applied to individual patients, not just
populations.
A good test for ruling in a condition has a positive likelihood
ratio of more than 10, meaning that if the patient has a positive
test, it is 10 times more likely that they have the disease as a
result of the positive test.
For example, if a very speci? c test, e.g. Troponin I, is positive,we know that myocardial damage has occurred, because the
number of false positives (B) is very low. Similarly, if a very
sensitive test, e.g. CT scan for abdominal aortic aneurysm (AAA),comes back as negative, we know that it is very unlikely that the
patient has an AAA as the number of false negatives (C) will be
very low.
However, Emergency Departments use many tests that are
not 100% sensitive or speci? c, and therefore a more powerful, but
less intuitive, model is necessary to understand these tests: likeli-
hood ratios , which are calculated from the speci? city and
sensitivity.
Treatment o rders
Once the likely diagnosis has been made, a set of treatment orders
needs to be decided and documented. A good acronym for this is
DAVID, e.g., for an elderly patient with an open fracture of the tibia:
· D iet – nil by mouth
· A ctivities – elevate limb
· V ital signs monitoring – hourly limb observations
· I nvestigations – CXR, FBC, U + E
· D rugstreatment – immediate i.v. antibiotics
Test result
Actual patient status (truth)
Disease present Disease absent
Positive True positive (A) False positive (B)
Negative False negative (C) True negative (D)
Total no.
patients
With disorder (A + C) Without disorder
(B + D)
Sensitivity = A(A + C) and Speci? city = D(B + D ) 14 Adult Emergency Medicine at a Glance, 1st edition. ? Thomas Hughes and Jaycen Cruickshank. Published 2011 by Blackwell Publishing Ltd.
3 Shock and i ntravenous ? uids
Cannula (actual size) Flow rate (mlmin)
Shock: causes
No pressure
Pump failurepump blockage Distribution failure Fluid failure
Cardiac failure PE
Cardiac tamponade
Septic shock
Anaphylaxis
Spinal shock
Wrong way
Blood loss, e.g. GI bleed
Penetrating trauma
300 mmHg
Pink 20G
Green 18G
Grey 16G
Orange 14G
50 mlmin
100 mlmin
200 mlmin
350 mlmin
100 mlmin
200 mlmin
400 mlmin
700 mlmin
Above 14G the diameter of the fluid delivery tube
may be the limiting factor
Intravenous ? uid therapy is a common medical treatment, but
recently there has been a reassessment of the role of intravenous
· uids as some of the hazards have become better understood.
Intravenous a ccess
Poiseulle s Law governing ? uid ? ow through a tube (assuming
laminar ? ow):
Flow
L
∝ Pr
4
η
where P = pressure difference, r = radius, η = viscosity, L = length.
Therefore the ideal resuscitation ? uid should be non - viscous,driven by pressure through a short, wide cannula. For resuscita-
tion, or when giving blood (viscous), a 16 G or larger cannula is
preferred. The large veins in the antecubital fossa and the femoral
vein are good for resuscitation but are prone to infection and
uncomfortable for patients in the long term. A pressure bag
in? ated to 300 mmHg doubles ? uid ? ow.
Before cannulation the skin should be thoroughly cleaned using
chlorhexidine in alcohol. A cannula in the forearm is (relatively)
comfortable for the patient and less likely to become infected
compared to other sites. Shock and intravenous ? uids 15
Special c ases
· Central lines are very useful in very sick patients, patients with
poor access or patients whose ? uid balance is particularly dif? cult
to regulate. Their length means that they are not ideal for deliver-
ing resuscitation ? uids. Introducer sheaths offer a large bore
central access.
· Intraosseus needles have to be drilled into adult bone to give
· uid, but can be life - saving. Bone marrow aspirate may be used
for blood cross - matching.
· Venous cut - down involves cutting the skin to be able to cannulate
a vein under direct vision. The long saphenous vein 1 cm above
and anterior to the medial malleolus, or the basilic vein in the
antecubital fossa, are the most common sites for this.
Types of i ntravenous ? uids
Crystalloids
Normal saline, Hartmann s solution and Ringer s lactate are solu-
tions that match plasma osmolality. All can be used to resuscitate
patients, and despite vigorous debate, no one variety has proven
superior clinical outcomes.
Dextrose
50% dextrose is used to resuscitate hypoglycaemic patients. 10%
dextrose is used to maintain a patient s blood sugar and prevent
hypoglycaemia, and 5% is used to give ‘ free water to avoid over-
loading with sodium or chloride.
Colloids
Colloids contain large molecules that help retain ? uid within
the intravascular space, which improves blood pressure in the
short term. Unfortunately these molecules leak out of damaged
capillaries, which may cause resistant oedema in the brain and
lungs, which increases mortality in head - injured patients. Colloids
may be helpful in sepsis, but should only be used by senior doctors.
Blood
A full cross - match takes 30 minutes but type - speci? c blood should
be available within minutes. If blood is needed before the blood
type is known, Group O Rhesus negative blood is used.
Whole blood as donated is the best substitute in trauma, but has
a short shelf life (days). Separating blood cells into ‘ packed cells
extends storage time to 3 months, but deterioration may mean that
the cells are not fully functional for 24 hours. The citrate used to
stabilise blood binds calcium ions, which can cause problems in
massive transfusions ( > 50% blood volume).
Fresh frozen plasma (FFP) or synthetic clotting factors can be
used to correct clotting problems. Tranexamic acid, platelets and
FFP are given as part of massive transfusion protocol.
Temperature
Evolution has given humans enzymes that function best at 37 ° C
and pH ≈ 7.4. Blood clotting is impaired in a cold acidotic patient,e.g. trauma patient. Temperature < 34 ° C and pH < 7.20 reduce
clotting to 1% of normal. Laboratory measurements at 37 ° C will
not accurately re? ect the clinical picture. For this reason, clotting
factors are given early in trauma resuscitation. Cold ? uids (4 ° C)
may be given after cardiac resuscitation as part of an active cooling
strategy to preserve brain function.
Shock
Shock is de? ned as inadequate tissue perfusion, i.e. not meeting
the metabolic demands of tissue. Pulse and blood pressure are
bedside measures of tissue perfusion, but are insensitive. pH, P CO 2 ,lactate and mixed venous blood oxygen levels, measured from a
central venous pressure (CVP) line, are better indicators.
Types of s hock
The body pumps a limited amount of ? uid around a series of
closed loops. Problems occur when the ? uid disappears, the pump
fails or the ? uid goes to the wrong loops.
Blood f ailure
Blood loss may be controlled or uncontrolled, internal or external.
Severe dehydration may cause similar problems.
Pump f ailure
The heart may fail due to internal pump problems, e.g. myocardial
infarction or heart failure, which impair the ability to pump.
Alternatively the pump may fail because there is in? ow obstruc-
tion (cardiac tamponade, tension pneumothorax) or out? ow
obstruction (pulmonary embolus, aortic dissection).
Distribution f ailure
Blood may be distributed to the wrong organs. Inappropriate
vasodilation occurs in septic shock, anaphylaxis and spinal shock
(due to loss of sympathetic tone below the injury) diverting blood
away from vital organs.
Grades of s hock
Compensated shock BP → HR ↑
Young adults are able to compensate for loss of blood volume by
vasoconstriction and increased cardiac output, maintaining a good
BP and perfusion of vital organs.
Decompensated shock BP ↓ HR ↑
The body s compensation mechanisms are overwhelmed, and the
blood pressure falls rapidly.
Fluid r esuscitation
Traditional t eaching: ‘ Fill ‘ e m u p
· Good blood pressure = good outcome.
· Poor blood pressure = poor outcome.
· Therefore give ? uidblood to achieve good blood pressure.
Unfortunately this is an oversimpli? cation. Short - term poor
perfusion is well tolerated and if blood loss has not been
controlled:
· ↑ blood pressure = ↑ blood loss.
Increased blood loss is due to loss of vasospasm, dilution of clot-
ting factors and dislodgement of clot.
Current t eaching: ‘ m inimal v olume ? uid r esuscitation
If there is uncontrolled bleeding (e.g. penetrating trauma, ruptured
AAA), large - bore intravenous access is obtained. The minimum
volume of ? uid necessary to maintain cerebral perfusion or a
systolic BP of 60 – 80 mmHg is used ( ‘ permissive hypotension ). The
priority is urgent control of bleeding in the operating theatre.
Exception: if there is brain injury, the need to maintain cerebral
perfusion pressure overrides hypotensive resuscitation. 16 Adult Emergency Medicine at a Glance, 1st edition. ? Thomas Hughes and Jaycen Cruickshank. Published 2011 by Blackwell Publishing Ltd.
4 Imaging in the Emergency Department
The FAST scan is performed to look for free abdominal fluid – usually blood.
The fluid tends to pool in certain areas within the peritoneal cavity
Focussed Assessment using Sonography in Trauma – FAST Scan
Fluid in pericardial sac ± tamponade
Recto-vesical pouch
Recto-uterine pouch Hepato-renal pouch
Cardiac view
Free fluid in the pelvis (requires full bladder)
Pelvic view
Normal – no fluid between kidney and spleen
Spleno-renal view
Normal
Positive
Shows fluid (blood) between liver
and kidney
Hepato-renal view (Morison’s Pouch)Imaging in the Emergency Department 17
Imaging use in the Emergency Department has increased rapidly
over the past few years due to technical advances and increasing
pressure to move decision - making earlier in a patient s journey,and to prevent unnecessary hospital admissions. Ultrasound is
now a core skill for senior Emergency Department doctors and
new hospitals often have a CT scanner in the Emergency
Department.
Plain r adiography
Plain radiographs interpreted by the treating clinician are used for
the majority of Emergency Department imaging. The advent of
digital radiography has made real - time reporting by radiologists
easier.
X - rays are ionising radiation and cause damage to tissues
through which they pass. The energy released is proportional to
the density of the tissue. Abdominal or thoracolumbar radio-
graphs should not be performed in young people, especially
females, without a very good reason, as the gonads are very radio-
sensitive. In this book, X - ray doses are expressed in terms of chest
radiographs (CXR). One CXR is approximately 3 days of back-
ground radiation.
X - rays are not therapeutic. If the result will not change manage-
ment, radiographs should not be taken. Examples include uncom-
plicated rib fractures (when not worried about a pneumothorax),coccyx pain and stubbed toes other than the big toe. Soft tissues
are poorly shown by plain ? lms, making it an insensitive examina-
tion for joints that rely on these for stability, e.g. knee, shoulder.
Reading p lain r adiographs
1 Check the patient s name and the date of the ? lm, particularly
on digital radiography systems, which offer many opportunities
for confusion.
2 There should be two good views of limbs: anterior - posterior and
lateral.
3 If requesting imaging of more than one area, ask yourself if this
is necessary. If not urgent, it may be better to re - examine the patient
once they have had some analgesia, or obtain a senior opinion.
4 You will learn more from your radiology department if you
engage with them and ask their advice rather than expecting a
purely technical service.
5 Many Emergency Departments operate a system whereby the
radiographer can ? ag an abnormality on the radiograph. You
should not dismiss something that the radiographer has ? agged as
abnormal without obtaining a senior opinion.
Clinical u ltrasound
Clinical (bedside) ultrasound use has increased exponentially with
the availability of cheap robust ultrasound machines, and is now
a core skill for Emergency Department doctors. Ultrasound has
been described as the ‘ visual stethoscope and is revolutionising
the assessment and management of patients in the Emergency
Department.
Ultrasound was initially used in the Emergency Department in
the resuscitation room for:
· Detecting abdominal aortic aneurysms (AAA).
· Focused abdominal scanning in trauma (FAST) scans, searching
for blood in the peritoneal cavity.
· Central venous line placement.
However, ultrasound use is now expanding to include:
· Shock assessment: cardiac function, vascular ? lling, signs of
pulmonary embolus, together with the AAA and FAST scans.
· Basic echocardiography.
· Deep vein thrombosis (DVT) scanning.
· Early pregnancy scanning.
· Hepato - biliary scanning.
Disadvantages are that ultrasound is operator dependent,requires training and skill validation, and can divert attention
from more important problems.
Computed t omography s can
As resolution and availability have increased and acquisition
times have dropped, computed tomography (CT) has become
an increasingly useful tool for the Emergency Department. CT is
very good for bony injuries, and the trauma CT has proved to
be more sensitive and speci? c than clinical examination in
major trauma, but requires a very large radiation dose (1000
CXR).
Neck imaging in high - risk trauma is routinely done by CT (100
CXR), as plain ? lms are insuf? ciently sensitive at detecting signi? -
cant injury. Examples of high - risk injuries are a high - speed rollo-
ver road traf? c collision, and also the elderly patient who falls
forward, hitting their face ( ‘ fall on outstretched face ), who is at
high risk of odontoid peg fracture, and in whom interpretation of
plain radiographs is very dif? cult (see Chapter 11 ).
Modern CT scanners have enough resolution and speed to be
able to resolve cardiac anatomy including the coronary arteries,pulmonary emboli and aortic dissection (400 CXR). CT brain scan
(100 CXR) is an essential part of the assessment of stroke or the
unconscious patient. CT KUB (kidneys, ureters and bladder; 400
CXRs) is the imaging of choice in renal colic.
Magnetic r esonance s can
Magnetic resonance (MR) scanning is rarely used in the Emergency
Department apart from possible cauda equina syndrome (acute
central disc prolapse pressing on the cauda equina), giving bowel
and bladder symptoms. MR scanning can be used to avoid the
large radiation dose incurred by CT, e.g. investigating renal colic
in young women.
Joints in which stability and function are mainly due to soft
tissues, i.e. ligaments and cartilage such as the knee and shoulder,are well imaged by MR scanning, but it is generally dif? cult to
access these directly from the Emergency Department.
Interventional i maging
Interventional imaging has an increasing role for a limited number
of severe conditions. Interventional imaging is generally offered
in larger hospitals, and together with trauma care, is one of the
main drivers for centralisation of acute services into large
hospitals.
· Primary percutaneous cardiac intervention with stenting has
become the treatment of choice for patients with myocardial
infarction.
· Endovascular treatments for patients with AAAs and aortic dis-
section are increasingly used. Neurosurgical bleeding from aneu-
rysms is treatable by coils, as is otherwise uncontrollable bleeding
in the pelvis, e.g. from pelvic fractures. 18 Adult Emergency Medicine at a Glance, 1st edition. ? Thomas Hughes and Jaycen Cruickshank. Published 2011 by Blackwell Publishing Ltd.
5 Analgesia
Intravenous regional anaesthesia
Non-pharmacological analgesia Analgesic ladder
Splint Plaster
Reduce dislocation
Sling, elevation,patient
positioning
Raise arm, inflate cuffs
↑300 mmHg
1 Inject prilocaine 2 Manipulate fracture,plaster, X-ray
3 Minimum 20 minutes later 4
↓0 mmHg
NSAID
Strong
Weak
Opiates
Ketorolac Morphine
Indometacin
Tramadol
Diclofenac
Ibuprofen Codeine
Paracetamol (acetaminophen)
Patients often arrive at the Emergency Department in pain, and
painkillers are often used before a de? nitive diagnosis is made.
This is humane, and enables a thorough examination to be per-
formed: there is no reason to withhold analgesia.
Patients are asked to rate the pain out of 10, with 0 being no
pain, and 10 being the worst pain they can imagine. This procedure
is repeated to gauge the effectiveness of the treatment and ensure
the pain is controlled.
In general, a patient s reported pain is taken at face value: ‘ pain
is what the patient feels and is treated as such. Patients seeking
opiates may fake pain, but this is rare.
Non - p harmacological a nalgesia
Splinting of fractures immobilises the bones, reducing pain. A
patient s anxiety and pain makes them tense, which may make
pain worse: a calm, supportive atmosphere and excellent nursing
care help to keep the patient relaxed.
Nitrous o xide
Nitrous oxide (N 2 O) combined with oxygen in a 1 : 1 mix in cylin-
ders (Entonox ? ) is often used, particularly out of hospital. It is a
short - term analgesic, effective only while the patient is breathing Analgesia 19
the gas, as it is rapidly cleared from the body. This ‘ laughing gas
is generally very safe, but should not be used in patients with a
possible pneumothorax.
Paracetamol ( a cetaminophen) and
c ompound a nalgesics
Paracetamol (acetaminophen) is effective and safe and can be
given orally, rectally or intravenously. Compound analgesics
consist of paracetamol combined with another analgesic, usually
low - dose codeine. They come in different strengths, the weaker of
which are sold without prescription. They are useful analgesics for
patients to be able to take home on discharge, but prescribing the
constituent drugs separately may allow more ? exibility.
Moderate o piates
· Codeine is a common component of compound analgesics, and
is effective but tends to cause constipation. Oxycodone and dihy-
drocodeine are more powerful variants of codeine, but offer little
extra bene? t, and have high abuse potential.
· Tramadol may be more effective than codeine. It has less abuse
potential than other drugs of comparable potency but should be
used with caution in the elderly.
Major o piates: m orphine, f entanyl,p ethidine ( m eperidine)
Opiates induce a feeling of well - being: patients, while still aware
of the pain, are not distressed by it. Young patients with major
fractures may require large doses of morphine, as will opiate
addicts who need analgesia. Intravenous opiates are used because
intramuscular absorption is unreliable and the intravenous route
enables analgesia to be titrated to response.
· Intravenous morphine is the gold standard of Emergency
Department analgesia. It is safe, predictable and effective.
Morphine is not as lipid soluble as other opiates, so does not give
a signi? cant ‘ high . Morphine often causes mild histamine release
that should not be confused with an allergic reaction. The duration
of action of morphine is approximately 3 hours.
· Fentanyl is a short - acting synthetic opiate that is particularly
useful when performing short procedures, as it is cleared from the
body within 30 minutes.
· Pethidine (meperidine) is quite lipid soluble and therefore sought
after by opiate addicts as it crosses the blood – brain barrier, giving
a ‘ high . It offers no bene? ts over morphine and should not be used
unless a patient has a de? nite allergy to morphine and there are
no other alternatives.
Non - s teroidal a nti - i n? ammatory d rugs
Injectable non - steroidal anti - in? ammatory drugs (NSAIDs), e.g.
ketorolac , are very effective in an Emergency Department setting.
They are particularly useful in patients with broken bones, colicky
pain (e.g. ureteric colic) and abdominal pain, but should be
avoided in elderly patients or those with active bleeding. An
equally effective alternative is a suppository (e.g. indometacin,diclofenac), which lasts for 16 hours.
Oral NSAIDs are useful as they can also be given to patients on
discharge. Ibuprofen is the least powerful, but has a relatively
benign side - effect pro? le.
Diclofenac and indometacin are more powerful NSAIDs but at
a cost of increased risk of side - effects.
Local a naesthesia and n erve b locks
· Lidocaine 1% is the local anaesthetic (LA) most often used for
wound management and is effective for 20 – 30 minutes without
adrenaline, or for 40 – 60 minutes with adrenaline.
· Adrenaline mixed with lidocaine increases length of action and
causes vasoconstriction giving a ‘ dry wound that is much easier
to assess, clean and close. Fear about using local anaesthetics with
adrenaline in digits was related to high concentrations (1 : 10 000);
less than 1 : 100 000 adrenaline is safe.
· Bupivicaine 0.25% is a long - acting local anaesthetic, lasting for
6 – 8 hours. Bupivicaine is highly protein bound: adrenaline does
not increase duration of action.
A safe maximum dose of lidocaine for wound in? ltration is 3 mg
kg, but with adrenaline is 6 mgkg. For bupivicaine the maximum
dose is 2 mgkg. Local anaesthetic toxicity ? rst causes perioral
parasthesia, and then ? ts and arrhythmias, and is treated by lipid
infusion.
Nerve blocks can offer very effective analgesia, e.g. digital and
femoral nerve blocks. Bupivicaine and lidocaine can be mixed to
provide a combination of rapid onset and long duration of action.
Local anaesthetic can also be injected into joints, e.g. for shoulder
dislocation.
A haematoma block can give good anaesthesia in minor frac-
tures e.g. Colle s fractures (Chapter 15 ). The skin is carefully
cleaned with alcohol and chlorhexidine and then up to 10 mL of
local anaesthetic is injected into the fracture haematoma. After
about 10 minutes reduction can be performed.
Intravenous r egional a naesthesia
(Bier s b lock)
Two intravenous cannulae are sited, one in the affected limb. A
double cuff is placed on the affected limb (usually the arm), which
is then lifted to exsanguinate it. The cuff is then in? ated well above
the systolic BP and local anaesthetic, e.g. prilocaine, injected.
Bupivicaine should never be used for intravenous regional
anaesthesia.
After waiting 5 minutes for the local anaesthetic to have maximal
effect, the operation, e.g. fracture reduction, is performed. The cuff
must not be de? ated until at least 20 minutes have elapsed from
injection of the local anaesthetic to avoid a bolus of undiluted local
anaesthetic perfusing the heart, potentially causing asystole. 20 Adult Emergency Medicine at a Glance, 1st edition. ? Thomas Hughes and Jaycen Cruickshank. Published 2011 by Blackwell Publishing Ltd.
6 Airway m anagement and s edation
Oropharyngeal airway
Measure from nose to tragus Measure from teeth to angle of jaw
Nasopharyngeal airway
Endotracheal tube in situ
Head tilt
Jaw thrust
Laryngoscope
Cricothyroid membrane
Endotracheal tube
View with a laryngoscope
Vocal cords
Epiglottis
Tongue
Needle cricothyroidotomy Surgical airway
Laryngoscope
Size 6 cuffed
endotracheal tube
Once the incision has been made NEVER let go
Check CO2
CXR
Oxygen tube
Three way tap with
extension tube
Cannula
Macintosh blade,size 3
8.0
7.0
Thyroid cartilage Cricothyroid
membrane
Cricoid
cartilage
Airway management in the Emergency Department is more chal-
lenging than in the operating room as patients presenting to the
Emergency Department must be assumed to be non - fasted, may
be physiologically unstable, and may have head, neck or facial
injuries.
Oxygenation and v entilation
Oxygenation is ensuring that the body has enough O 2 ; ventilation
is ensuring that there is suf? cient air? ow to remove CO 2 . Oxygen
consumption is markedly increased in the acutely unwell patient,and giving high concentrations of oxygen supports the metabolic
demands of the body in acute illness. However, high levels of
oxygen may paradoxically make some ischaemic injury worse, e.g.,brain heart due to vasoconstriction. A normal ‘ Hudson O 2 mask
can give inspired oxygen (FiO 2 ) concentrations of up to 60%. They
should not be used with O 2 < 4 Lmin to prevent CO 2 build - up. A
mask with a reservoir bag or a self - in? ating bag - valve - mask can
increase FiO 2 to about 90% with high ? ow ( > 10 Lmin O 2 ). A Venturi mask gives accurate low FiO 2 e.g. 28%. Nasal prongs give
a variable amount of O 2 approx 25 – 30% but should only be used
with low ? ow rates (2 Lmin O 2 ).
Ventilatory f ailure
Under normal circumstances, an increased level of CO 2 is the main
driver to breathe. Patients with chronic lung disease, usually
COPD, become immune to this drive. For these ‘ blue bloater
patients, a low blood O 2 level drives breathing: their CO 2 level will
be high.
If high FiO 2 is given to these patients, it reduces their respiratory
drive, increasing their CO 2 levels further, making them sleepy,which further decreases their drive to breathe, etc. An oxygen satu-
ration target of 91% in these patients balances the need for tissue
oxygenation against that for ventilation.
· O 2 should be prescribed, with a target saturation
· A patient with O 2 sat > 96% probably does not need extra O2 unless
high metabolic need, e.g., sepsis, trauma
· If FiO 2 has given a very high CO 2 level, reduce FiO 2 slowly
· If in doubt, give O 2 and obtain a senior review
Suction
A Yankauer suction catheter is used to suction blood, vomit or
secretions in the oropharynx. To avoid causing the patient to
vomit, do not suction the oropharynx if the patient is conscious,and ‘ only suck where you can see .
Airway s upport
The jaw thrust, head tilt, oropharyngeal and nasopharangeal airways
are illustrated opposite . The oropharyngeal airway is sized as the
distance between the patient s teeth and the angle of the mandible.
The nasopharyngeal airway should be the same length as the distance
between the tip of the nose and the tragus of the ear.
Laryngeal m ask a irway
Emergency Department patients are not fasted and the laryngeal
mask airway (LMA) does not prevent stomach contents being
aspirated, nor can high ventilation pressures be achieved, as might
be necessary in asthmatic patients. For these reasons the LMA is
not a ‘ de? nitive airway and is not normally used in the Emergency
Department.
Endotracheal t ube
The most common means to provide a de? nitive airway, the endotra-
cheal tube (ETT), is a plastic tube that is inserted through the mouth
(or rarely the nose) into the trachea. There is a cuff that is in? ated to
seal against the tracheal mucosa, and a radio - opaque line to indicate
position on X - ray. The ETT should be secured, e.g. with tape, and
the position checked by CO 2 monitoring and a chest X - ray.
Endotracheal tubes are sized by their internal diameter: 7.0 mm
for an adult female, 8.0 mm for a male. There are markings indicat-
ing distance from the tip: this is to avoid the tube being pushed
WARNING
Airway management and sedation 21
too far, e.g. down the right main bronchus, which is larger and
straighter than the left.
The decision that the patient needs intubation is the responsibil-
ity of the doctor managing the airway. Factors indicating need for
intubation include:
· Airway instability: bleeding into airways, airway burns.
· Coma: Glasgow Coma Scale (GCS) < 9, deteriorating level of
consciousness, loss of protective laryngeal re? exes.
· Inadequate oxygenation: despite high inspired O 2 (FiO 2 ).
· Inadequate ventilation: patient tireddrowsy.
· Therapeutic reasons: control seizures, hypothermia.
· Pragmatic reasons: combative patient, need for transport.
A laryngoscope is needed to insert the ETT. In some countries,straight (Miller) blades are used; in others, curved (Macintosh)
blades. These have a light to enable sight of the larynx.
McGill s forceps have a ‘ kink in them to avoid the operator s
hands obstructing the ? eld of vision. They are useful for removing
loose items in the oropharynx, and manipulating the ETT.
Surgical a irway
Rarely, a situation occurs when it is not possible to intubate or
ventilate a patient. In this situation, there are two options:
· A needle cricothyroidotomy will provide short - term oxygenation,but is not a de? nitive airway, and CO 2 levels will build up.
· A surgical airway through the cricoid membrane using a 6.0 mm
cuffed ETT provides a de? nitive airway.
Procedural s edation
Procedural sedation is often performed in the Emergency
Department to allow relocation of dislocations or for short painful
procedures. The person performing the sedation needs appropriate
skills and experience to manage any potential situation, including
the need for intubation.
The procedure should be carried out in a resuscitation bay with
full monitoring, oxygen and suction equipment. Two doctors
should be present at all times to ensure that the doctor administer-
ing the sedation has their full attention on the patient s airway.
The patient should be fasted for at least 4 hours, should give
formal consent, and the doctor should stay with ......
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