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P16基因甲基化状态与子宫肌瘤发生机制的研究(2)
http://www.100md.com 2011年8月1日 石彩歌 方晓萍 肖志奇
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    [1] Bodner-Adler B, Bodner K, Czerwenka K, Kimberger O,Leodolter S, Mayerhofer K (2010) Expression of p16 protein in patients with uterine smooth muscle tumors: an immunohistochemic alanalysis. Gynecol Oncol 96:62-66

    [2] O'Neill CJ, McBrie HA, Connolly LE, McCluggage WG (2009)Uterine leiomyosarcomas are characterized by high p16, p53 andMIB1 expression in comparison with usual leiomyomas, leiomyoma variants and smooth muscle tumours of uncertain m alignant potential. Histopathology 50:851-858

    [3] Cook JD, Walker CL. Treatment strategies for uterine leiomyoma:the role of hormonal modulation [J]. Semin Reprod Med, 2009, 22: 105- 111

    [4] Maruo T,Ohara N,Wang J, et a1 Sex steroidal regulation of uterine leiomyoma growth and apoptosis[J].Hum Reprod Update,2009,1O(3):207-220

    [5] Sakaguchi H,Fujimoto J, Aoki I, eta1.Expression of estrogen receptor alpha and beta in myometrium of premenopausal and postmenopausal women[J].Steroids,2010,68(1):11-19

    [6]Mitchell S.Rein, W.Lee Powell, Frederick C.Walters al .Cytogenetic abnormalities in uterine myomas are associated with myoma sizeMolecular Human Reproduction vol.4 no.1 pp. 83-86, 2009

    作者单位: 广东省珠海市妇幼保健院

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