调心方对APP/PS1双转基因AD小鼠空间学习记忆能力和海马突触传递长时程增强的影响(4)
[3] 宗园媛,王晓映,王海林,等.APP/PS1双转基因阿尔茨海默病小鼠模型的老年斑及行为学动态分析[J].中国比较医学杂志,2008,18(9):8-12.
[4] KORTE M, HERRMANH U, ZHANG X, et al. The role of APP and APLP for synaptic transmission, plasticity and network function:lessons from genetic mouse models[J]. Exp Brain Res,2012,217(3/4):435-440.
[5] TAKASHIMA S, IESHIMA A. Dementia and the down syndrome[J]. Brain Develop,1989,11:131-133.
[6] MOOLMAN D L, VITOLO O V. Dendrite and dendritic spine alterations in Alzheimer models[J]. J Neurocytol,2004,33:377-387.
[7] GONG B. Ubiquitin hydrolase Uch-L1 rescues beta-amyloid-induced decreases in synaptic function and contextual memory[J]. Cell, 2006,126:775-788.
[8] DONNA L, SMIT H. Reversal of long-term dendritic spine alteration in Alzheimer disease models[J]. PNAS,2009,39:16877-16882.
[9] SCHMID A W, LYNCH M A, HERRON C E. The effects of IL-1 receptor antagonist on beta amyloid mediated depression of LTP in the rat CA1 in vivo[J]. Hippocampus,2009,19(7):670-676.
(收稿日期:2016-12-19)
(修回日期:2017-02-15;編辑:华强), 百拇医药(王晓雯 邓海燕 王健 徐颖)
[4] KORTE M, HERRMANH U, ZHANG X, et al. The role of APP and APLP for synaptic transmission, plasticity and network function:lessons from genetic mouse models[J]. Exp Brain Res,2012,217(3/4):435-440.
[5] TAKASHIMA S, IESHIMA A. Dementia and the down syndrome[J]. Brain Develop,1989,11:131-133.
[6] MOOLMAN D L, VITOLO O V. Dendrite and dendritic spine alterations in Alzheimer models[J]. J Neurocytol,2004,33:377-387.
[7] GONG B. Ubiquitin hydrolase Uch-L1 rescues beta-amyloid-induced decreases in synaptic function and contextual memory[J]. Cell, 2006,126:775-788.
[8] DONNA L, SMIT H. Reversal of long-term dendritic spine alteration in Alzheimer disease models[J]. PNAS,2009,39:16877-16882.
[9] SCHMID A W, LYNCH M A, HERRON C E. The effects of IL-1 receptor antagonist on beta amyloid mediated depression of LTP in the rat CA1 in vivo[J]. Hippocampus,2009,19(7):670-676.
(收稿日期:2016-12-19)
(修回日期:2017-02-15;編辑:华强), 百拇医药(王晓雯 邓海燕 王健 徐颖)