Th17在RSV感染婴幼儿的表达上调及其可能的临床意义(2)
 |
| 第1页 |
参见附件。
[2]Lukens MV，van de Pol AC，Coenjaerts FE， et al.A systemic neutrophil response precedes robust CD8(t) T-cell activation during naturalrespiratory syncytial virus infection in infants[J].J Virol，2010，84：2374-2383.
[3]Mukherjee S，Lindell DM，Berlin AA，et al.IL-17-induced pulmonary pathogenesis during respiratory viral infection and exacerbation of allergic disease[J].Am J Pathol 2011，179：248-258.
[4]Stoppelenburg AJ，Salimi V，Hennus M，et al.Local IL-17A potentiates early neutrophil recruitment to the respiratory tract during severe RSV infection[J].PLoS One，2013，8：e78461.
[5]Weaver C T，Harrington LE，Mangan PR，et al.Th17： an effector CD4 T cell lineage with regulatory T cell ties[J].Immunity，2006；24(6)：677-688.
[6]Hashimoto T，Akiyama K，Kobayashi N，et al.Comparison of IL-17 production by helper T cells among atopic and nonatopic asthmatics and control subjects[J].Int Arch Allergy Immunol，2005，137(Suppl1)：51-54.
[7]Linden A，Hoshino H，Laan M.Airway neutrophils and interleukin-17[J].Eur Respir J，2000，15(5)：973-977.
[8]de Roock S，Stoppelenburg AJ，Scholman R，et al.Defective TH17 development in human neonatal T cells involves reduced RORC2 mRNA content[J].J Allergy Clin Immunol，2013，132(3)：754-756
编辑/哈涛
您现在查看是摘要介绍页，详见PDF附件。