当前位置: 首页 > 医疗版 > 疾病专题 > 皮肤科 > 色素障碍性皮肤病(色素沉着) > 白癜风
编号:13114530
白癜风皮损浸润T细胞亚群的研究进展(3)
http://www.100md.com 2017年5月28日 《医学信息》 2017年第21期
     [4]Sandoval-Cruz,Garcia-Carrasco,Sanchez-Porras,et al.Immunopathogenesis of vitiligo[J].Autoimmunity Reviews,2011,10(12):762-765.

    [5]JG van,D Konijnenberg,TA Dellemijn,et al.Autoimmune destruction of skin melanocytes by perilesional T cells from vitiligo patients[J].Journal of Investigative Dermatology,2009,129(9):2220-2232.

    [6]Antelo DP,Filgueira AL,Cunha JM.Reduction of skin-homing cytotoxic T cells(CD8+ -CLA+)in patients with vitiligo[J].Photoder-matol Photoimmunol Photomed,2011,27(1):40-44.
, http://www.100md.com
    [7]Palermo B,Campanelli R,Garbelli S,et al.Specific cytotoxic Tlymphocyte responsesagainst Melan-AMART1,tyrosinase and gp100 in vitiligo by the use of major histocopatibility complexpeptide tetramers:the role of cellular immunity in the etiopathoge-gesis of vitiligo[J].Invest Dermatol,2001,117(2):326-332.

    [8]Mandelcorn-Monson RL,Shear NH,Yau E,et al.Cytotoxic T lymphocyte reactivity to gp100,Melan AMART-1,and tyrosinase,in HLA-A2-positive vitiligo patients[J].Journal of Investigative Dermatology,2003(121):550-556.
, 百拇医药
    [9]Lang KS,Caroli CC,Muhm A,et al.HLA-A2 restricted,melanocyte-specific CD8+T lymphocytes detected in vitiligo patients are related to disease activity and are predominantly directed against Melan AMART-1[J].Journal of Investigative Dermatology,2001(116):891-897.

    [10]EH Kemp,NG Gavalas,DJ Gawkrodger,et al.Autoantibody responses to melanocytes in the depigmenting skin disease vitiligo[J].Autoimmunity Reviews,2007,6(3):138-142.

, http://www.100md.com     [11]K Ongenae,GN Van,JM Naeyaert.Evidence for an autoimmune pathogenesis of vitiligo[J].Pigment Cell&Melanoma Research,2003,16(2):90-100.

    [12]Wańkowicz-Kalińska A,van RM,Tigges BJ,et al.Immuno-polarization of CD4+and CD8+T cells to Type-1-like is associated with melanocyte loss in human vitiligo[J].Laboratory investigation,2003,83(5):683-695.

    [13]NH Kim,D Torchia,P Rouhani,et al.Tumor necrosis factor-α in vitiligo:direct correlation between tissue levels and clinical parameters[J].Cutaneous&Ocular Toxicology,2011,30(3):225-227.
, 百拇医药
    [14]Lv Y,Li Q,Wang L,et al.Use of anti-tumor necrosis factor agents:apossible therapy for vitiligo[J].Medical Hypotheses,2009(72):546-547.

    [15]Galadari I.Serum levels of the soluble interleukin-2 receptor in vitiligo patients in UAE[J].Eur Ann Allergy&Clin Immunol,2005(37):109-111.

    [16]Franczuk A,Szepietowski JC,Noworolska A.Serum concentrationsof interleukin-2 soluble receptor(IL-2 sR)in patients with vitiligo:relationship with type and extent of the disease[J].Acta Dermatovenerologica Croatica,2004(12):71-76.
, http://www.100md.com
    [17]Y Kotobuki,A Tanemura,L Yang,et al.Dysregulation of me-lanocyte function by Th17-related cytokines:significance of Th17 cell infiltration in autoimmune vitiligo vulgaris[J].Pigment Cell Melanoma Res,2012,25(2):219-230.

    [18]DA Bassiouny,O Shaker.Role of interleukin-17 in the pathogenesis of vitiligo[J].Clinical and Experimental Dermatology,2011,36(3):292-297.

    [19]Wang CQ,Cruz-Inigo AE,Fuentes-Duculan J,et al.Th17 cells and activated dendritic cells are increased in vitiligo lesions[J].PLoS One,2011(6):e18907.

    [20]Fontenot,Rasmussen,P Jeffrey,et al.Regulatory T cell lineage specification by the forkhead transcription factor foxp3[J].Immunity,2005,22(3):329-341.編辑/成森, http://www.100md.com(戴远洋 宋鹏飞 王京)
上一页1 2 3


    参见:首页 > 医疗版 > 疾病专题 > 皮肤科 > 色素障碍性皮肤病(色素沉着) > 白癜风