蜕皮甾酮对氧化损伤的人晶状体上皮细胞凋亡的防护作用(4)
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[2] Spector A. The lens and oxidative stress. In: Sies Hed. Oxidative stress, oxidants and antioxidadants [M]. London: Academic Press, 1991:529-558.
[3] Delcourt C, Cristol JP, Leger CL, et al.Associations of antioxidant enzymes with cataract and age-related macular degeneration. The POLA Study[J]. Pathologies Oculaires Liees a l'Age. Ophthalmology, 1999, 106(2): 215-222.
[4] Li WC, Jerome R, Wang GM, et al. Calcimycin-induced lens epithelial cell apoptosis contributes to cataract formation [J]. Ex. Eye Res, 1995, 61: 91-98.
[5] Li WC,Kuszak JR,Dunn K,et al.Lens epithelial cell apoptosis appears to be a common cellular basis for non-congenital cataract development in humans and animals[J].Biol,1995,130 (1):169-181.
[6] Wang X, Simpkins JW, Dykens JA,et a1.Oxidative damage to human lens epithelial cells in Culture:estrogen protection of mitochondrial potential, ATP,and cell viability [J].Invest Ophthalmol Vis Sci,2003,44(5):2067-2075.
[7] Moor AN, Flynn JM, Gottipati S.17β-estradiol stimulates MAPK signaling pathway in human lens epithelial cell cultures preventing collapse of mitochondrial membrane potential during acute oxidative stress[J]. Mitochondrion,2005,5(4):235-247.
[8] Moor AN,Gottipati S,Mallet RT, et al. A putative mitochondrial mechanism for antioxidative cytoprotection by 17 betaestradiol[J].Exp Eye Res,2004,78:933-944.
[9] 赵丕文,牛建昭,王继峰,等.蜕皮甾酮和蜕皮甾酮对人乳腺癌T47D细胞增殖及ER亚型表达的影响[J].北京中医药大学学报,2007,30(4):242-245.
[10] Kerr JFR,Wyllie All,Currie AR.Apoptosis:a basic biological phenonlenon with wide-ranging implications in tissuckinetics[J].BR JCancer,1972,26(4):239-257.
[11] 彭黎明.六种细胞凋亡检测方法比较[J].中华病理杂志,1999,28(1):55-58.
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