当前位置: 首页 > 期刊 > 《医药产业资讯》 > 2008年第12期 > 正文
编号:11606159
自身免疫性脑脊髓炎大鼠CD4+CD25+T细胞、CD86分子的变化及意义(2)
http://www.100md.com 2008年6月22日 焦景亚 魏晓阳
    参见附件(634kb)。

     [参考文献]

    [1]Sun Y,Qiao J,Lu CZ,et al. Increase of circulating CD4+CD25+T cells in myasthemia gravis patients with stability and thymectomy[J].Clin Immunol,2004,112(3):284-289.

    [2]Bugen L,Dallmon S.Co-stimulation of T cells[J]. Respir Cur Canc Med,2000,162(4):164-168.

    [3]Encinas J A,Lees MB,So bel RA, et al. Identication of genetic loci associate with paralysis, inflammation and weight loss in mouse experimental autoimmune encephalomyelitis[J].Int Immunol,2001,13(3):257-264.

    [4]Hafler DA.Loss of functional suppression by CD4+CD25+ Regulatory T cells in patients with multipul sclerosis[J].J Exp Med,2004,199(7):971-979.

    [5]Sto A,Naka jima H,lkeda K,et al.CD4+CD25+Tcell development is regulatea by at least distinct mechanisms[J].Blood, 2002,99:555-560.

    [6]Kukreja.Multiple immune regulatory defects in type-1 diabetes[J].J Clinical invest,2002,109: 131-140.

    [7]朱晓勇,李大金,孙晓溪, 等.大鼠抗小鼠CD80/CD86单克隆抗体的制备与鉴定[J].上海免疫杂志,2001,21(1):55-56.

    (收稿日期:2008-01-03)

    注:本文中所涉及到的图表、注解、公式等内容请以PDF格式阅读原文

您现在查看是摘要介绍页,详见PDF附件(634kb)