贝那普利对原发性高血压C反应蛋白水平的影响及其临床意义(2)
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[4]Grau AJ, Buggle F, Becher H, et al. The assoclation of leucocyte count fibroinogen and C reactive protein with vascular risk factors and ischemic vascular diseases[J].Thromb Res,1996,(82):245.
[5]Ichtlscherer S, Rosenherer G, Dirk H, et al. Elevated C reactive protein levels and impaired endothelial vasoreactivity in patients with coronary artery disease[J].Circulation,2000,102(13):1000.
[6]Bhagat K, Vallance P. Inflammatory cytokines impair endothelium dependent dilation in human veins in vivo [J].Circulation,1997,96(1):3042.
[7]Yasunari K, Maeda K, Nakamura M, et al. Oxidative stress in leukocytes is a possible link between blood glucose, and C reactive protein[J].Hypertension,2002,39(1):777.
[8]Hennandedeesa MA, Bustos C, Ortego M, et al. ACE Inhibitior quinapril reduces the arterial expression NF-кB dependent prom flam matory factors but not collagen I in a rabbit model of atheroscle rosis [J].Am J Parhol,1998,(153):1825-1837.
[9]黄怀宇,陈文煜.细胞粘附因子与动脉粥样硬化的关系[J].中国微循环,2002,6(3):187-189.
[10]Da Cunha V,Tharn DM,Martin-McNulty B, et al. Enalapril attenuates angiotensin Ⅱ-induced atheroscle rosis and vascular in flam mation[J].A theroscle rosis,2005,178(1):9-17.
(收稿日期:2010-02-01)
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