寻常型银屑病皮损中磷酸化信号转导和转录激活因子—3与白介素—17的表达及意义(3)
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[13] Zaba LC,Cardinale I,Gilleaudeau P,et al. Amelioration of epidermal hyperplasia by TNF inhibition is associated with reduced Th17 responses [J]. J Exp Med,2007,204(13):3183-3194.
[14] 罗迪青,赵玉昆,刘隽华,等.Th17细胞、IL-17A mRNA及IL-23R mRNA与寻常型银屑病关系研究[J].中华皮肤科杂志,2011,44(12):879-881.
[15] Yang XO,Pappu BP,Nurieva R,et al. T helper 17 lineage differentiation is programmed by orphan nuclear receptors ROR alpha and ROR gamma [J]. Immunity,2008,28(1):29-39.
[16] Al Khatib S,Keles S,GarciaLloret M,et al. Defects along the Th17 differentiation pathway underlie genetically distinct forms of the hyperige syndrome [J]. J Allergy Clin Immunol,2009,124(2):342-348.
[17] Kimura A,Naka T,Kishimoto T. IL-6-depenednt and –independent pathways in the development of interleukin 17-producing T helper cells [J]. Proc Natl Acad Sci USA,2007,104(29):12099-120104.
[18] Mangan PR,Harrington LE,O′Quinn DB,et al. Transforming growth factorbeta induces development of the T(H)17 lineage [J]. Nature,2006,441(7090):231-234.
[19] Zhou L,Spolski R,Min R,et al. IL-6 programs T(H)-17 cell differentiation by promoting sequential engagement of the IL-21 and IL-23 pathways [J]. Nat Immunol,2007,8(9):967-974.
[20] Yang F,Wu WF,Yan YL,et al. Expression of il-23/th17 pathway in a murine model of coxsackie virus b3-induced viral myocarditis [J]. Virology Journal,2011,8:301-309.
(收稿日期:2012-04-17 本文编辑:谷俊英)
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