铅对星形胶质细胞影响的研究进展(2)
[3]Hansson E,Zugner R.Can chronic pain and spreading of pain be induced via glial mechanisms New hypotheses on the generators maintaining protracted pain conditions[J].Lakartidningen,2005,102(47):3552～3558.
[4]Jia QH,Jiang JJ,Ha XQ.Toxic Effect of Lead Acetate on HFAEC and Astrocytes[J].Occup and H ealth,2010,26:626～629.
[5]Zhang Y,Sun LG,Ye LP,et al.Lead-induced stress response in endoplasmic reticulum of astrocytes in CNS[J].Toxicol Mech Methods,2008,18(9):751～757.
[6]Gonzalez-Gronow M,Selim MA,Papalas J,et al.GRP78:a multifunctional receptor on the cell surface[J].Antioxid Redox Signal,2009,11(9):2299～2306.
[7]White LD,Cory-Slechta DA,Gilbert ME,et al.New and evolving concepts in the Neurotoxicology of lead[J].ScienceDirect Toxicology and Applied Phamacology,2007,225(1):1～27.
[8]Struzyska L,Dabrowska-Bouta B,Koza K,et al.Inflammation-Like Glial Response in Lead-Exposed Immature Rat Brain[J].Toxicological Sciences,2007,95(1):156～162.
[9]Deborah W,Florence C,Barbara B,et a1.Lead exposure and attention regulation in children living in poverty[J].Developmental Medicine and Child Neurology,2004,46(12):825～832.
[10]Schousboe A.Role of astrocytes in the maintenance and modulation of glutamatergic and GABAergic neurotransmission[J].Neurochem.Res,2003,28(2):347～352.
[11]Rose CR,Blum R,Pichler B,et al.Truncated TrkB-T1 mediates neurotrophin-evoked calcium signaling in glia cells[J].Nature,2003,426(6962):74～78.
[12]Zhang Y,Ye LP,Wang B,et al.Effect of lead on ERK activity and the protective function of bFGF in rat primary culture astroglia[J].J Zhejiang Univ Sci B,2007,8(6):422～427.
[13]Birnbanm S.G,Varga A.W,Yuan L.L,et a1.Structure and function of Kv4-family transient potassium channels[J].Physiol Rev,2004,84(3):803～833.
【收稿日期】 2011-03-09, http://www.100md.com(李珺 韩杰 唐玲荣)
[4]Jia QH,Jiang JJ,Ha XQ.Toxic Effect of Lead Acetate on HFAEC and Astrocytes[J].Occup and H ealth,2010,26:626～629.
[5]Zhang Y,Sun LG,Ye LP,et al.Lead-induced stress response in endoplasmic reticulum of astrocytes in CNS[J].Toxicol Mech Methods,2008,18(9):751～757.
[6]Gonzalez-Gronow M,Selim MA,Papalas J,et al.GRP78:a multifunctional receptor on the cell surface[J].Antioxid Redox Signal,2009,11(9):2299～2306.
[7]White LD,Cory-Slechta DA,Gilbert ME,et al.New and evolving concepts in the Neurotoxicology of lead[J].ScienceDirect Toxicology and Applied Phamacology,2007,225(1):1～27.
[8]Struzyska L,Dabrowska-Bouta B,Koza K,et al.Inflammation-Like Glial Response in Lead-Exposed Immature Rat Brain[J].Toxicological Sciences,2007,95(1):156～162.
[9]Deborah W,Florence C,Barbara B,et a1.Lead exposure and attention regulation in children living in poverty[J].Developmental Medicine and Child Neurology,2004,46(12):825～832.
[10]Schousboe A.Role of astrocytes in the maintenance and modulation of glutamatergic and GABAergic neurotransmission[J].Neurochem.Res,2003,28(2):347～352.
[11]Rose CR,Blum R,Pichler B,et al.Truncated TrkB-T1 mediates neurotrophin-evoked calcium signaling in glia cells[J].Nature,2003,426(6962):74～78.
[12]Zhang Y,Ye LP,Wang B,et al.Effect of lead on ERK activity and the protective function of bFGF in rat primary culture astroglia[J].J Zhejiang Univ Sci B,2007,8(6):422～427.
[13]Birnbanm S.G,Varga A.W,Yuan L.L,et a1.Structure and function of Kv4-family transient potassium channels[J].Physiol Rev,2004,84(3):803～833.
【收稿日期】 2011-03-09, http://www.100md.com(李珺 韩杰 唐玲荣)