表皮极性及极性蛋白在银屑病中研究进展(4)
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参见附件。
[19]Ebnet K. Organization of multiprotein complexes at cell-cell junctions[J]. Histochem Cell Biol, 2008, 130(1): 1-20.
[20] Hu Y J, Wang Y D, Tan F Q, et al. Regulation of paracellular permeability: factors and mechanisms[J]. Mol Biol Rep, 2013, 40(11): 6123-6142.
[21] Itoh M, Furuse M, Morita K, et al. Direct binding of three tight junction-associated MAGUKs, ZO-1, ZO-2, and ZO-3, with the COOH termini of claudins[J]. J Cell Biol, 1999, 147(6): 1351-1363.
[22]Furuse M, Hata M, Furuse K, et al. Claudin-based tight junctions are crucial for the mammalian epidermal barrier: a lesson from claudin-1-deficient mice[J]. J Cell Biol, 2002, 156(6): 1099-1111.
[23]Troy T C, Rahbar R, Arabzadeh A, et al. Delayed epidermal permeability barrier formation and hair follicle aberrations in Inv-Cldn6 mice[J]. Mech Dev, 2005, 122(6): 805-819.
[24]Gumbiner B M. Regulation of cadherin-mediated adhesion in morphogenesis[J]. Nat Rev Mol Cell Biol, 2005, 6(8): 622-634.
[25]Philippova M, Pfaff D, Kyriakakis E, et al. T-cadherin loss promotes experimental metastasis of squamous cell carcinoma[J]. Eur J Cancer, 2013, 49(8): 2048-2058.
[26]Young P, Boussadia O, Halfter H, et al. E-cadherin controls adherens junctions in the epidermis and the renewal of hair follicles[J]. EMBO J, 2003, 22(21): 5723-5733.
[27]Aurrand-Lions M, Johnson-Leger C, Wong C,et al. Heterogeneity of endothelial junctions is reflected by differential expression and specific subcellular localization of the three JAM family members[J]. Blood, 2001, 98(13): 3699-3707.
[28]Griffiths C E, Barker J N. Pathogenesis and clinical features of psoriasis[J]. Lancet, 2007, 370(9583): 263-271.
(收稿日期:2013-11-11) (本文编辑:黄新珍)
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