辛伐他汀对阿霉素致心衰兔心功能的影响(3)
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参见附件。
[4] Field K M.Effect of 3-Hydroxy-3-methylglutaryl coenzyme a reductase inhibitors on high-sensitivity C-reactive protein levels[J].Pharmacotherapy：The Journal of Human Pharmacology and Drug Therapy，2005，25(10)：1365-1377.
[5]顾复生，朱文玲，戴闺柱，等.慢性收缩性的心力衰竭治疗建议[J].中华心血管病杂志，2002，30(1)：7.
[6] Nissen S E，Tuzcu E M，Schoenhagen P，et al.Statin therapy，LDL cholesterol，C-reactive protein and coronary artery disease[J].N Engl J Med，2005，352(1)：29-38.
[7] Ridker P M，Cannon C P，Morrow D，et al.C-reactive protein levels and outcomes after statin therapy[J].N Ensl J Med，2005，352(1)：20-28.
[8] Yoon Y W，Kwon H M，Hwang K C，et al.Upstream regulation of matrix metalloproteinase by EMMPRIN；extracellular matrix metalloproteinase inducer in advanced atherosclerotic plaque[J].Atherosclerosis，2005，180(1)：37-44.
[9] Bonnet J，McPherson R，Tedgui A，et al.Comparative effects of 10 mg versus 80 mg Atorvastatin on high-sensitivity C-reactive protein in patients with stable coronary artery disease：results of the CAP(Comparative Atorvastatin Pleiotropic effects) study[J].Clin Ther，2008，30(12)：2298-2313.
[10] Liu J，Shen Q，Wu Y.Simvastatin prevents cardiac hypertrophy in vitro and in vivo via JAK/STAT pathway[J].Life Sci，2008，82(19)：991-996.
[11] Wu L J，Zhao L Y，Zheng Q S，et al.Simvastatin attenuates hypertrophic responses induced by cardiotrophin-1 via JAK-STAT pathway in cultured cardiomyocytes[J].Molecular and Cellular Biochemistry，2006，284(1-2)：65-71.
(收稿日期：2013-10-31) (本文编辑：欧丽)
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