中电导钙激活钾离子通道(SK4)的研究进展(3)
[15]Wang, Z.H., et al., Blockage of intermediate-conductance-Ca2(+)-activated K+ channels inhibits progression of human endometrial cancer. Oncogene, 2007. 26(35): p. 5107-5114.
[16]Reich, E.P., et al., Blocking ion channel KCNN4 alleviates the symptoms of experimental autoimmune encephalomyelitis in mice. European Journal of Immunology, 2005. 35(4): p. 1027-36.
[17]Grgic, I., et al., Blockade of T-lymphocyte KCa3.1 and Kv1.3 channels as novel immunosuppression strategy to prevent kidney allograft rejection. Transplant Proc, 2009. 41(6): p. 2601-6.
, 百拇医药
[18]Di, L., et al., Inhibition of the K+ channel KCa3.1 ameliorates T cell-mediated colitis. Proc Natl Acad Sci U S A, 2010. 107(4): p. 1541-6.
[19]Toldi, G., et al., Lymphocyte calcium influx characteristics and their modulation by Kv1.3 and IKCa1 channel inhibitors in healthy pregnancy and preeclampsia. Am J Reprod Immunol, 2011. 65(2): p. 154-63.
[20]Mahaut-Smith, M.P. and L.C. Schlichter, Ca2(+)-activated K+ channels in human B lymphocytes and rat thymocytes. J Physiol, 1989. 415: p. 69-83.
, 百拇医药
[21]Moritoki, Y., et al., AMA production in primary biliary cirrhosis is promoted by the TLR9 ligand CpG and suppressed by potassium channel blockers. Hepatology, 2007. 45(2): p. 314-22.
[22]Hickman-Davis, J.M., et al., Killing of Klebsiella pneumoniae by human alveolar macrophages. Am J Physiol Lung Cell Mol Physiol, 2002. 282(5): p. L944-56.
[23]Gao, Y.D., et al., Calcium-activated K(+) channel (K(Ca)3.1) activity during Ca(2+) store depletion and store-operated Ca(2+) entry in human macrophages. Cell Calcium, 2010. 48(1): p. 19-27.
, 百拇医药
[24]Valverde, P., T. Kawai, and M.A. Taubman, Potassium channel-blockers as therapeutic agents to interfere with bone resorption of periodontal disease. J Dent Res, 2005. 84(6): p. 488-99.
[25]Tharp, D.L., et al., Upregulation of intermediate-conductance Ca2+-activated K+ channel (IKCa1) mediates phenotypic modulation of coronary smooth muscle. American Journal of Physiology-Heart and Circulatory Physiology, 2006. 291(5): p. H2493-H2503.
, 百拇医药
[26]Toyama, K., et al., The intermediate-conductance calcium-activated potassium channel KCa3.1 contributes to atherogenesis in mice and humans. Journal of Clinical Investigation, 2008. 118(9): p. 3025-3037.
[27]Cipolla, M.J., et al., SKCa and IKCa Channels, Myogenic Tone, and Vasodilator Responses in Middle Cerebral Arteries and Parenchymal Arterioles Effect of Ischemia and Reperfusion. Stroke, 2009. 40(4): p. 1451-1457.
[28]Crane, G.J., et al., Small- and intermediate-conductance calcium-activated K+ channels provide different facets of endothelium-dependent hyperpolarization in rat mesenteric artery. Journal of Physiology-London, 2003. 553(1): p. 183-189., 百拇医药(张汝超)
[16]Reich, E.P., et al., Blocking ion channel KCNN4 alleviates the symptoms of experimental autoimmune encephalomyelitis in mice. European Journal of Immunology, 2005. 35(4): p. 1027-36.
[17]Grgic, I., et al., Blockade of T-lymphocyte KCa3.1 and Kv1.3 channels as novel immunosuppression strategy to prevent kidney allograft rejection. Transplant Proc, 2009. 41(6): p. 2601-6.
, 百拇医药
[18]Di, L., et al., Inhibition of the K+ channel KCa3.1 ameliorates T cell-mediated colitis. Proc Natl Acad Sci U S A, 2010. 107(4): p. 1541-6.
[19]Toldi, G., et al., Lymphocyte calcium influx characteristics and their modulation by Kv1.3 and IKCa1 channel inhibitors in healthy pregnancy and preeclampsia. Am J Reprod Immunol, 2011. 65(2): p. 154-63.
[20]Mahaut-Smith, M.P. and L.C. Schlichter, Ca2(+)-activated K+ channels in human B lymphocytes and rat thymocytes. J Physiol, 1989. 415: p. 69-83.
, 百拇医药
[21]Moritoki, Y., et al., AMA production in primary biliary cirrhosis is promoted by the TLR9 ligand CpG and suppressed by potassium channel blockers. Hepatology, 2007. 45(2): p. 314-22.
[22]Hickman-Davis, J.M., et al., Killing of Klebsiella pneumoniae by human alveolar macrophages. Am J Physiol Lung Cell Mol Physiol, 2002. 282(5): p. L944-56.
[23]Gao, Y.D., et al., Calcium-activated K(+) channel (K(Ca)3.1) activity during Ca(2+) store depletion and store-operated Ca(2+) entry in human macrophages. Cell Calcium, 2010. 48(1): p. 19-27.
, 百拇医药
[24]Valverde, P., T. Kawai, and M.A. Taubman, Potassium channel-blockers as therapeutic agents to interfere with bone resorption of periodontal disease. J Dent Res, 2005. 84(6): p. 488-99.
[25]Tharp, D.L., et al., Upregulation of intermediate-conductance Ca2+-activated K+ channel (IKCa1) mediates phenotypic modulation of coronary smooth muscle. American Journal of Physiology-Heart and Circulatory Physiology, 2006. 291(5): p. H2493-H2503.
, 百拇医药
[26]Toyama, K., et al., The intermediate-conductance calcium-activated potassium channel KCa3.1 contributes to atherogenesis in mice and humans. Journal of Clinical Investigation, 2008. 118(9): p. 3025-3037.
[27]Cipolla, M.J., et al., SKCa and IKCa Channels, Myogenic Tone, and Vasodilator Responses in Middle Cerebral Arteries and Parenchymal Arterioles Effect of Ischemia and Reperfusion. Stroke, 2009. 40(4): p. 1451-1457.
[28]Crane, G.J., et al., Small- and intermediate-conductance calcium-activated K+ channels provide different facets of endothelium-dependent hyperpolarization in rat mesenteric artery. Journal of Physiology-London, 2003. 553(1): p. 183-189., 百拇医药(张汝超)