《循环研究杂志》.2005年.第5期
文章精
- Dysfunctional Smad Signaling Contributes to Abnormal Smooth Muscle Cell Proliferation in Familial Pulmonary Arterial Hypertension
- p38 Mitogen-Activated Protein Kinase Activates eNOS in Endothelial Cells by an Estrogen Receptor -Dependent Pathway in Response to Black Tea
- Cardiac Sarcoplasmic Reticulum Calcium Release and Load Are Enhanced by Subcellular cAMP Elevations in PI3K-Deficient Mice
- Cardiac Overexpression of Melusin Protects From Dilated Cardiomyopathy Due to Long-Standing Pressure Overload
- Oxygen Regulation of Tumor Perfusion by S-Nitrosohemoglobin Reveals a Pressor Activity of Nitric Oxide
- Myocardial Interstitial Matrix Metalloproteinase Activity Is Altered by Mechanical Changes in LV Load
- Bidirectional Ventricular Tachycardia and Fibrillation Elicited in a Knock-In Mouse Model Carrier of a Mutation in the Cardiac Ryanodine Rec
- Functional Characterization of Connexin43 Mutations Found in Patients With Oculodentodigital Dysplasia
- Angiogenesis and Pericytes in the Initiation of Ectopic Calcification
- Interdependency of -Adrenergic Receptors and CFTR in Regulation of Alveolar Active Na+ Transport
- Adenovirus-Mediated Overexpression of O-GlcNAcase Improves Contractile Function in the Diabetic Heart
- Reprogramming of the Human Atrial Transcriptome in Permanent Atrial Fibrillation
- Adipose Tissue, Inflammation, and Cardiovascular Disease
- Endotoxin-Induced Myocardial Dysfunction
- Proapoptotic Effects of Caspase-1/Interleukin-Converting Enzyme Dominate in Myocardial Ischemia
- Adenovirus Vector E4 Gene Regulates Connexin 40 and 43 Expression in Endothelial Cells via PKA and PI3K Signal Pathways
- Angiotensin IV Activates the Nuclear Transcription Factor-B and Related Proinflammatory Genes in Vascular Smooth Muscle Cells
- Regulation of Vascular Smooth Muscle Cell Proliferation
- Novel Mitogenic Effect of Adenosine on Coronary Artery Smooth Muscle Cells
- Role of Histamine Produced by Bone MarroweCDerived Vascular Cells in Pathogenesis of Atherosclerosis
- Modulation of the Cardiac Sodium Channel NaV1.5 by Fyn, a Src Family Tyrosine Kinase
- Long-Term Inhibition of RhoA Attenuates Vascular Contractility by Enhancing Endothelial NO Production in an Intact Rabbit Mesenteric Artery
- Cell-to-Cell Connection of Endothelial Progenitor Cells With Cardiac Myocytes by Nanotubes
- Sequelae of Insulin-Resistant Adipose Tissue
- Angiotensin II Enhances Interleukin-18 Mediated Inflammatory Gene Expression in Vascular Smooth Muscle Cells